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RDCS Pediatric Echocardiography Cheat Sheet

Normal Anatomy & Physiology

15%of exam

Segmental Situs & Ventricular LoopFetal Shunts & Postnatal ClosureCoronary Ostia & Arch SidednessPLSVC & Conotruncal Embryology

Acquired Heart Disease

12%of exam

Kawasaki Coronary Z-Score CutoffsDCM vs HCM FeaturesEndocarditis & Pulmonary HypertensionCardiac Tamponade & Myocarditis

Congenital Anomalies

25%of exam

ASD, VSD, PDA & CAVSDTOF, D-TGA & L-TGACoA, HLHS & TAPVR TypesEbstein, Truncus & ALCAPA

Postoperative Anatomy

19%of exam

BTT Shunt vs Sano ConduitNorwood, Glenn & FontanJatene ASO vs Mustard/SenningRastelli, Ross & Repaired TOF

Performing the Exam

29%of exam

Bernoulli, Qp:Qs & RVSPPediatric Z-Scores & WindowsLV SF, RV TAPSE & TeiDoppler Angle & Bubble Contrast

Quick Facts

Exam
ARDMS Pediatric Echocardiography (PE)
Credential
RDCS (requires SPI + PE)
Questions
~145 MCQ and hotspot items
Time Limit
3-hour examination period
Pass Score
555 on 300-700 scale
Initial Credential Timing
Calendar-based ARDMS five-year rule
Pass Rate
88% first-time (2023 ARDMS)
Retake Wait
≥60 days; eligible later PE window

Morphologic RV vs Morphologic LV

Morphologic Right Ventricle

  • Apically offset trileaflet tricuspid valve
  • Septal chordal attachments + moderator band
  • Coarse trabeculations + muscular conus

Morphologic Left Ventricle

  • Basal bileaflet mitral valve
  • No septal chordal attachments
  • Finer trabeculations; evaluate complete connections

Integrate morphology; position alone is insufficient

Segmental Situs & Fetal Circulation

Situs Solitus Abdominal Pattern
Aorta left; IVC right/anterior
Right Atrial Isomerism
Bilateral right atrial appendage morphology
Left Atrial Isomerism
Bilateral left atrial appendage morphology
Morphologic RV Markers
Moderator band; coarse trabeculations; tricuspid valve
Three Fetal Shunts
Ductus venosus, foramen ovale, ductus arteriosus
PLSVC Clue
Dilated coronary sinus; trace venous drainage

Coronaries, Arch Sidedness & Embryology

Coronary Origins
Trace ostia and proximal courses
Coronary Dominance
Defined by posterior descending artery supply
Arch Sidedness
Trace arch relative to trachea
Common Arch Branching Variant
Shared brachiocephalic/left carotid origin
Conotruncal Development
Outflow alignment and septation are complex
AV Septal Defect Morphology
Common AV junction; variable bridging leaflets

AHA Kawasaki Z-Score Thresholds

Z thresholds: 2 dilation; 2.5 small; 5 medium; 10 giant

<2: No current involvement2 to <2.5: Dilation2.5 to <5: Small5 to <10 AND <8mm: Medium≥10 OR ≥8mm: Giant

Pediatric DCM vs Pediatric HCM

Dilated Cardiomyopathy (DCM)

  • Ventricular dilation with systolic dysfunction
  • Assess size using appropriate pediatric models
  • Secondary mitral regurgitation may occur

Hypertrophic Cardiomyopathy (HCM)

  • Hypertrophy beyond expected loading conditions
  • Obstructive and nonobstructive phenotypes exist
  • SAM/dagger jet only when applicable

Dilation/dysfunction vs inappropriate myocardial hypertrophy

AHA 2024 Current Coronary Classification

  1. Coronary Z <2→No current coronary involvement
  2. Coronary Z ≥2 and <2.5→Dilation only
  3. Coronary Z ≥2.5 and <5→Small aneurysm
  4. Z ≥5, <10 AND diameter <8mm→Medium aneurysm
  5. Z ≥10 OR diameter ≥8mm→Large/giant aneurysm

Kawasaki Z-Scores & Pediatric Acquired

Kawasaki Small Aneurysm
Z ≥2.5 and <5
Kawasaki Medium Aneurysm
Z ≥5, <10 AND diameter <8mm
Kawasaki Giant Aneurysm
Z ≥10 OR diameter ≥8mm
Endocarditis Vegetation Pattern
Often upstream; negative echo cannot exclude
Pediatric Pulmonary Hypertension Clues
Septal flattening; contextual Doppler pressure estimates
Tamponade Collapse Pattern
RA systolic; RV early diastolic collapse

Tetralogy of Fallot Four Components

PROVe: Pulmonary obstruction, RV hypertrophy, Overriding aorta, VSD

P: Pulmonary/infundibular stenosisR: Right ventricular hypertrophyO: Overriding aorta; assess connectionsVe: Large malaligned VSD

Common D-TGA vs ccTGA Connections

D-TGA (Simple Transposition)

  • AV concordance + VA discordance
  • Parallel circulations; cyanotic neonate
  • Often anterior/rightward aorta; variants exist

ccTGA (Double Discordance)

  • Double discordance (AV and VA)
  • Physiologic series flow; systemic RV
  • Associated VSD, PS, Ebstein, CHB

VA discordance vs double discordance

ASD, VSD, PDA & CAVSD

Sinus Venosus Defect
Caval junction; assess pulmonary venous connections
Significant Atrial Left-to-Right Shunt
May cause RA/RV volume loading
Outlet VSD
Assess adjacent aortic cusp support
PDA Imaging
Trace duct; interpret directional Doppler
Complete AV Septal Defect
Common AV junction; atrial/ventricular communications
Rastelli AVSD Types A/C
A: crest-attached; C: free bridging leaflet

Darling Four Types of TAPVR

I Supracardiac, II Cardiac, III Infracardiac, IV Mixed drainage

I Supracardiac: above-heart drainageII Cardiac: Coronary sinus or RAIII Infracardiac: below-heart drainageIV Mixed: Multiple drainage levels

ALCAPA vs AAOCA Coronary Anomalies

ALCAPA (Bland-White-Garland)

  • LCA arises from main PA
  • Infant DCM + bright papillary muscle
  • Septal collaterals + retrograde PA flow

AAOCA (Opposite Sinus)

  • Coronary arises from opposite sinus
  • Course may be interarterial/intramural
  • Risk depends on detailed anatomy

Pulmonary origin vs opposite aortic sinus

TOF, TGA, CoA, HLHS & TAPVR

TOF Defining Pattern
Malalignment VSD, overriding aorta, RVOT obstruction
TOF Coronary Check
Identify coronary branches crossing RVOT
Coarctation Assessment
Anatomy plus flow; low output masks
HLHS Spectrum
Underdeveloped left heart; assess systemic pathway
Infracardiac TAPVR
Below-diaphragm drainage; frequent obstruction
Ebstein Supportive Displacement
≥8mm/m² supportive; assess complete morphology

Truncus Arteriosus & Coronary Anomalies

Truncus Type I
Short main pulmonary artery then bifurcation
Truncus Types II/III
Separate branch origins: posterior/lateral patterns
Truncus Associations
Assess arch; consider 22q11.2 deletion
ALCAPA Clues
LV dysfunction, MR, bright papillary muscles
ALCAPA Flow
May show retrograde LCA-to-PA flow
AAOCA Risk Features
Assess ostium, interarterial and intramural course

Three-Stage Single-Ventricle Palliation

Stage 1 Norwood, Stage 2 Glenn, Stage 3 Fontan completion

1 Norwood: Neoaorta + BTT/Sano2 Glenn: SVC to RPA3 Fontan: IVC to PA

Modified BTT Shunt vs Sano Conduit

Modified BTT Shunt

  • Innominate/subclavian artery to PA
  • Continuous systole-diastole forward flow
  • May produce diastolic systemic runoff

Sano Conduit

  • Systemic RV infundibulum to PA
  • To-and-fro systolic/diastolic Doppler
  • Avoids direct systemic arterial shunt runoff

Systemic artery source vs RV source

Postoperative Anatomy Assessment

  1. Known arterial switch repair→Assess transferred coronaries and both outflows
  2. Known Rastelli repair→Assess LV tunnel and RV–PA conduit
  3. Historic Mustard/Senning baffle→Assess venous baffles and systemic RV
  4. Known Ross repair→Assess autograft and RV–PA conduit
  5. Single-ventricle staged palliation→Identify actual systemic/pulmonary venous pathways

Norwood, Glenn & Fontan Palliation

Norwood Palliation
Systemic neoaorta plus pulmonary flow source
Bidirectional Glenn
SVC connects to pulmonary arterial circulation
Glenn Doppler
Assess venous phasicity and focal acceleration
Fontan Completion
Systemic venous return bypasses subpulmonary ventricle
Fontan Fenestration
Usually venous-to-atrial decompression; assess gradient
Glenn Pulmonary AV Malformations
May cause delayed pulmonary bubble transit

Jatene ASO, Rastelli, Ross & TOF

Arterial Switch LeCompte Maneuver
Pulmonary bifurcation positioned anterior to aorta
Arterial Switch Follow-up
Coronaries, outflows, neoaortic valve and root
Mustard/Senning Baffles
Trace venous pathways; assess systemic RV
Rastelli Anatomy
LV-to-aorta tunnel plus RV-to-PA conduit
Ross Procedure
Pulmonary autograft replaces aortic valve
Repaired TOF
Quantify PR, obstruction and RV response

Intracardiac Shunt vs Isolated PDA Flow

ASD or VSD (L-to-R)

  • Shunt enters upstream of PV
  • RVOT stroke volume > LVOT
  • Loading depends on shunt magnitude
  • Assumes no significant semilunar regurgitation

Isolated PDA (L-to-R)

  • Shunt enters distal to PV
  • LVOT stroke volume > RVOT
  • Significant shunt loads LA/LV
  • Assumes no significant semilunar regurgitation

RVOT exceeds LVOT vs opposite

Shunt Pattern Interpretation

  1. Suspected apical atrial septal dropout→Confirm anatomy/flow from additional windows
  2. Left-arm contrast fills coronary sinus→Evaluate PLSVC and coronary sinus drainage
  3. Agitated saline: LA within 3–6 cycles→Likely interatrial shunt; confirm pathway(Count cardiac cycles after RA opacification)
  4. Agitated saline: LA after >6 cycles→Likely pulmonary transit; confirm circulation(Count cardiac cycles after RA opacification)
  5. Isolated left-to-right PDA→LVOT flow can exceed RVOT flow

Bernoulli, Qp:Qs & Pressure Calculations

Simplified Bernoulli
ΔP = 4v²; negligible proximal velocity
Non-negligible Proximal Velocity
Consider 4(v₂²−v₁²); verify assumptions
Stroke Volume
Area × VTI; area = πd²/4
Shunt Flow Ratio
Define sampling sites and shunt anatomy
VSD-Derived RVSP
SBP − 4v²; appropriate L→R assumptions
PASP With RVOT Obstruction
RVSP minus simultaneous RV-to-PA pressure drop

Measurement Method and Assumptions

  1. Proximal velocity negligible; assumptions satisfied→Estimate peak instantaneous gradient: 4v²
  2. Proximal velocity not negligible→Assess expanded Bernoulli and lesion geometry
  3. Distorted LV invalidates linear shortening→Use suitable volumetric assessment
  4. Complex or systemic RV anatomy→Integrate multiple suitable function indices
  5. PW aliasing limits velocity measurement→Adjust PRF or use appropriate CW

Z-Scores, Views & Ventricular Function

Pediatric Z-Score
Match method, model and body size
Pediatric Aortic Root
Inner edges; peak-flow midsystolic measurement
LV Shortening Fraction
(LVIDd − LVIDs) / LVIDd ×100%
RV Function Assessment
Integrate age-adjusted and geometry-sensitive indices
Tei Index
(IVCT + IVRT) / ejection time
Pulmonary Venous Assessment
Trace connections using multiple windows

Common Traps

Dropout vs True Septal Defect

Septal dropout ≠ confirmed ASD ≠ Multiview anatomy ≠ isolated dropout artifact

PDA vs Intracardiac Flow Sampling

PDA entry ≠ upstream pulmonary-valve entry ≠ PDA Qp:Qs ≠ RVOT÷LVOT

Pediatric vs Adult Root Convention

Pediatric inner-edges ≠ traditional adult leading-edges ≠ Z-score model convention ≠ interchangeable methods

Outlet VSD and Cusp Support

Outlet VSD ≠ guaranteed aortic regurgitation ≠ Cusp prolapse ≠ preserved cusp support

TOF Coronary Variants

LAD crossing RVOT ≠ normal course ≠ Normal ostium ≠ confirmed proximal course

Doppler vs Catheter Gradients

Maximum instantaneous ≠ catheter peak-to-peak ≠ Peak fenestration gradient ≠ measured PVR

Last Minute

  1. 1.PE: ≈145 items; 3 hours
  2. 2.Passing scaled score: 555 on 300–700
  3. 3.Five-year deadline uses calendar years
  4. 4.Kawasaki giant: Z≥10 OR diameter≥8mm
  5. 5.Medium: Z≥5,<10 AND diameter<8mm
  6. 6.Ebstein ≥8mm/m² supports morphology
  7. 7.Subcostal or parasternal protocol starts
  8. 8.Root: inner-edge peak-flow midsystole
  9. 9.RVOT diameter: end diastole
  10. 10.LV shortening fraction depends on geometry
  11. 11.RV adult cutoffs need pediatric context
  12. 12.MPI = (IVCT+IVRT)/ET
  13. 13.Bubble timing depends on circulation
  14. 14.Agitated saline detects right-to-left shunt pathways
  15. 15.Manufactured microbubbles normally cross pulmonary circulation
  16. 16.PW baseline shift preserves Nyquist limit
  17. 17.Doppler alignment affects squared gradient
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