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Key Facts: UKOM Sp.KJ Exam

Not published

Official CBT item count

Kolegium Psikiatri Indonesia National CBT structure

Not published

Official CBT duration

CBT Nasional PPDS Psikiatri examination schedule

Not published

Passing standard

Official sources reviewed

Perkonsil 104/2021

Curricular Standard

Standar Pendidikan Profesi Dokter Spesialis Kedokteran Jiwa (KKI)

Not published

National cohort pass rate

National residency exit examination statistics

CBT confirmed

Current format established by sources reviewed

National exit assessment framework for Sp.KJ

Independent UKOM Sp.KJ practice by OpenExamPrep. A current national CBT for Indonesian psychiatry residents is confirmed, while its item count, duration, pass standard and any additional components were not published in the sources reviewed. This 100-question English-language MCQ bank supports independent knowledge review and is not an official translation or testing simulation.

Sample UKOM Sp.KJ Practice Questions

Try these sample questions to review concepts for the UKOM Sp.KJ exam. Each question includes a detailed explanation. Start the interactive quiz above for the full 100+ question experience with AI tutoring.

1A 24-year-old male is brought to the psychiatric emergency department by his family because he has been isolating himself in his bedroom for the past seven months. Over the last two months, he has exhibited prominent persecutory delusions that neighbors are broadcasting his thoughts via satellite, third-person auditory hallucinations commenting on his actions, and marked emotional blunting with poverty of speech. Comprehensive laboratory testing and brain MRI reveal no organic etiology, and urine toxicology is negative. According to PPDGJ-III (ICD-10) and DSM-5 diagnostic criteria, which diagnosis is MOST appropriate?
A.Schizophreniform disorder
B.Schizophrenia
C.Brief psychotic disorder
D.Delusional disorder
Explanation: The patient meets diagnostic criteria for schizophrenia under both PPDGJ-III/ICD-10 (F20) and DSM-5. The clinical presentation features Schneiderian first-rank symptoms (thought broadcasting and running commentary hallucinations) along with negative symptoms (emotional blunting, alogia) persisting for greater than 6 months total duration, with at least 1 month of active-phase psychotic symptoms.
2A 31-year-old woman has had an uninterrupted three-year illness with auditory hallucinations and persecutory delusions. Major depressive episodes meeting full criteria occupied most of the total illness duration, but hallucinations and delusions also persisted for at least six months when no major mood episode was present. Which diagnosis is most accurate?
A.Major depressive disorder with psychotic features
B.Bipolar I disorder, current episode depressed, severe with psychotic features
C.Schizoaffective disorder, depressive type
D.Schizotypal personality disorder
Explanation: Schizoaffective disorder requires both a major mood episode concurrent with the schizophrenia syndrome and at least two weeks of delusions or hallucinations without a major mood episode. Mood episodes must also be present for the majority of the total active and residual illness duration. The vignette now supplies both longitudinal requirements.
3A 48-year-old civil servant believes firmly that his superior at work has placed covert surveillance devices in his car and office to undermine his career. This conviction has been present for 8 months. Mental status examination reveals well-organized persecutory beliefs, clear sensorium, coherent speech, and fully intact affect. He has never experienced hallucinations, his hygiene is immaculate, and his general daily functioning and performance outside of this specific conflict remain unaffected. What is the MOST appropriate diagnosis according to PPDGJ-III (F22)?
A.Paranoid schizophrenia (F20.0)
B.Persistent delusional disorder (F22.0)
C.Schizoid personality disorder (F60.1)
D.Generalized anxiety disorder (F41.1)
Explanation: Persistent delusional disorder (gangguan waham menetap / F22.0 in PPDGJ-III) is characterized by the development of a single delusion or a set of related delusions that are usually persistent and sometimes lifelong, lasting at least 1 month, in the absence of prominent hallucinations, affective flattening, or formal thought disorder. Social and occupational functioning outside the direct impact of the delusion is preserved.
4A 22-year-old university student with no prior psychiatric history experiences the sudden onset of intense persecutory delusions, disorganized speech, and erratic agitated behavior 4 days after failing an important national licensing exam. Physical exam, head CT, and drug screen are normal. She is admitted to the psychiatric ward and treated with low-dose risperidone. By day 18 of admission, all psychotic symptoms have completely resolved, and she demonstrates full insight and returns to her baseline premorbid level of functioning. What is the correct diagnosis?
A.Brief psychotic disorder (F23)
B.Schizophrenia (F20)
C.Adjustment disorder with disturbance of conduct (F43.2)
D.Bipolar I disorder with psychotic features (F31)
Explanation: Brief psychotic disorder (gangguan psikotik akut dan sementara / F23 in PPDGJ-III) involves the acute onset of at least one positive psychotic symptom (delusions, hallucinations, disorganized speech, or grossly disorganized behavior) with a duration of at least 1 day but less than 1 month, followed by an eventual full return to premorbid functioning.
5A 28-year-old man diagnosed with chronic schizophrenia presents in an immobile, unresponsive state. On examination, he remains completely silent despite vigorous verbal stimulation, maintains an uncomfortable awkward arm posture against gravity for over an hour when positioned by the examiner, and resists passive movement with a force proportional to the examiner's effort (gegenhalten). What specific catatonic sign is demonstrated when the patient's limbs maintain positions imposed by the examiner?
A.Echopraxia
B.Waxy flexibility (flexibilitas cerea)
C.Stereotypy
D.Automatic obedience
Explanation: Waxy flexibility (flexibilitas cerea / catalepsy) is a pathognomonic catatonic sign where the patient offers initial resistance before allowing their limbs to be molded into postures, which are then maintained for prolonged periods against gravity.
6A 34-year-old female presents with acute catatonia characterized by mutism, stupor, refusal to eat or drink for 48 hours, and marked negativism. Vital signs are normal. What is the FIRST-LINE diagnostic and therapeutic intervention of choice?
A.Intramuscular haloperidol 5 mg
B.Intravenous lorazepam 1 to 2 mg challenge
C.Oral clozapine 25 mg
D.Immediate intravenous dantrolene 1 mg/kg
Explanation: The lorazepam challenge test (1 to 2 mg IV or IM) is the first-line diagnostic and therapeutic gold standard for catatonia. Marked clinical improvement within 30 to 60 minutes confirms catatonia and guides scheduled lorazepam dosing (often 6 to 12 mg/day). First-generation antipsychotics like haloperidol must be strictly avoided because dopamine antagonism can precipitate malignant catatonia or neuroleptic malignant syndrome.
7A 29-year-old male with catatonic stupor fails to improve after 72 hours of scheduled intravenous lorazepam at 12 mg/day. He develops a core temperature of 39.2°C, blood pressure of 170/105 mmHg, heart rate of 128 bpm, diaphoresis, and profound lead-pipe muscular rigidity. Serum creatine kinase (CK) is elevated at 14,500 U/L. What is the MOST definitive, life-saving psychiatric intervention?
A.Switching to oral olanzapine 20 mg daily
B.Intravenous valproate loading
C.Emergency electroconvulsive therapy (ECT)
D.High-dose intramuscular fluphenazine
Explanation: This patient has developed malignant catatonia / neuroleptic-refractory catatonia, characterized by autonomic instability, hyperthermia, and extreme rigidity. Emergency Electroconvulsive Therapy (ECT) is the definitive, life-saving gold-standard treatment with response rates exceeding 80-90% when benzodiazepines fail.
8What is the primary neurochemical mechanism responsible for the therapeutic antipsychotic efficacy of first-generation antipsychotics (FGAs) such as haloperidol?
A.Serotonin 5-HT2A receptor antagonism in the prefrontal cortex
B.Dopamine D2 receptor antagonism in the mesolimbic pathway
C.Dopamine D2 receptor agonism in the tuberoinfundibular tract
D.Gamma-aminobutyric acid (GABA-A) positive allosteric modulation
Explanation: The therapeutic efficacy of first-generation antipsychotics in treating positive psychotic symptoms (delusions, hallucinations) stems primarily from high-affinity antagonism of dopamine D2 receptors in the mesolimbic pathway, where hyperdopaminergic signaling underlies positive symptoms.
9Compared to first-generation antipsychotics, second-generation (atypical) antipsychotics such as risperidone and olanzapine exhibit a substantially lower propensity to induce extrapyramidal symptoms (EPS). Which pharmacological property accounts MOST for this reduced EPS liability?
A.Selective blockade of histamine H1 receptors
B.Potent antagonism of 5-HT2A receptors relative to D2 receptors
C.Competitive inhibition of monoamine oxidase A (MAO-A)
D.Stimulation of central alpha-1 adrenergic receptors
Explanation: Second-generation antipsychotics are characterized pharmacologically by potent serotonin 5-HT2A receptor antagonism combined with D2 receptor antagonism. Serotonin normally inhibits dopamine release in the nigrostriatal pathway; 5-HT2A blockade disinhibits dopamine release in the striatum, competing with D2 blockade and significantly reducing the incidence of extrapyramidal symptoms.
10Aripiprazole is categorized as a third-generation antipsychotic with a unique pharmacological profile. Which receptor mechanism best describes its primary action at dopamine D2 receptors?
A.Pure competitive antagonist
B.Partial agonist
C.Irreversible inverse agonist
D.Allosteric potentiator
Explanation: Aripiprazole functions as a partial agonist at dopamine D2 receptors (with intrinsic activity around 25-30%). In hyperdopaminergic environments (e.g., mesolimbic tract in psychosis), it acts functionally as an antagonist, reducing positive symptoms. In hypodopaminergic environments (e.g., mesocortical and nigrostriatal tracts), it provides intrinsic baseline dopaminergic tone, reducing EPS and negative symptom liability.

About the UKOM Sp.KJ Exam

The current national CBT is a specialty competency examination for eligible psychiatry residents in Indonesia. Perkonsil No. 104 Tahun 2021 supplies published education and competency scope; additional current components were not established by the official sources reviewed.

Exam sponsor: Kolegium Kedokteran Jiwa Indonesia / KKI. The requirements and fees below concern the certification or admission exam, separate from our free practice resources.

Assessment

Question count not published by the exam provider

Time Limit

Not published in the official sources reviewed.

Passing Score

Not published in the official sources reviewed.

Exam / Certification Fees

Not published in the official sources reviewed.

Exam sponsor website

Reported exam pass rate: Not published in the official sources reviewed.. Not published in the official sources reviewed. Exam sponsor website

Fees, eligibility, and exam policies can change. Confirm them with the exam sponsor before applying or paying.

Our practice resources: topics covered

We aim to reflect publicly available exam outlines and topic information in our study resources. Coverage, format, and difficulty may differ from the actual exam, and we cannot guarantee that every detail is accurate or current. Confirm exam requirements, fees, and policies with the official exam sponsor.

25%

Schizophrenia Spectrum & Psychotic Disorders

Diagnosis under PPDGJ-III and DSM-5, schizoaffective disorder, delusional disorder, catatonia management (lorazepam challenge and ECT), FGA vs SGA pharmacology, EPS, tardive dyskinesia, metabolic monitoring, and clozapine hematologic protocols.

25%

Mood & Anxiety/Stress-Related Disorders

Major depressive disorder, melancholic vs atypical features, suicide safety planning, bipolar I vs II, acute mania management, bipolar depression, lithium monitoring and toxicity, valproate/lamotrigine safety, GAD, panic disorder, OCD ERP protocols, and PTSD.

20%

Neurocognitive & Substance Use Disorders

Delirium criteria and motor subtypes, Alzheimer disease, vascular dementia, Lewy body dementia, frontotemporal dementia, alcohol withdrawal (CIWA-Ar), Wernicke-Korsakoff encephalopathy, opioid overdose and MAT, and stimulant-induced psychosis.

15%

Child, Adolescent & Geriatric Psychiatry

ADHD psychostimulants, autism spectrum disorder irritability, oppositional defiant vs conduct disorder, Tourette disorder, adolescent depression (fluoxetine safety), pseudodementia vs true dementia, BPSD protocols, and Beers criteria.

15%

Consultation-Liaison, Forensic Psychiatry, Psychotherapy & Emergencies

Neuroleptic malignant syndrome, serotonin syndrome, emergency agitation de-escalation, ECT indications, Visum et Repertum Psikiatrik (VeRP), KUHP Pasal 44 criminal responsibility, UU Kesehatan No. 17/2023, CBT models, and medical ethics.

Preparing for the UKOM Sp.KJ Exam

What You Need to Know

  • Passing score: Not published in the official sources reviewed.
  • Assessment: Question count not published by the exam provider
  • Time limit: Not published in the official sources reviewed.
  • Exam / certification fees: Not published in the official sources reviewed. Official sources

Using Our Practice Resources

  • Work through all 100 available questions
  • Review every answer and explanation
  • Track weak areas and revisit them
  • Use our AI tutor for tough concepts

UKOM Sp.KJ: Suggested Study Strategy

1Master differential diagnostic criteria between primary psychotic disorders and mood disorders with psychotic features under both PPDGJ-III and DSM-5.
2Know psychopharmacology monitoring parameters thoroughly: clozapine ANC cutoffs, lithium therapeutic windows (0.8-1.2 mEq/L acute, 0.6-0.8 mEq/L maintenance), and SGA metabolic screening intervals.
3Distinguish psychiatric emergencies quickly: NMS (lead-pipe rigidity, hyporeflexia, extreme CK) versus Serotonin Syndrome (clonus, hyperreflexia, agitation).
4Understand the legal basis of Indonesian forensic psychiatry: Visum et Repertum Psikiatrik (VeRP) under KUHAP Pasal 120/133, criminal responsibility under KUHP Pasal 44, and involuntary emergency criteria under UU Kesehatan No. 17/2023.
5Differentiate delirium motor subtypes, prioritizing recognition of hypoactive delirium which carries high mortality and is frequently misdiagnosed as depression.
6Review pediatric and geriatric pharmacotherapy nuances: fluoxetine as the first-line antidepressant in youth, and Beers criteria avoiding high anticholinergic burden in the elderly.

Frequently Asked Questions

What is the UKOM Sp.KJ examination?

The UKOM Sp.KJ is the national specialty competency examination conducted by Kolegium Psikiatri Indonesia for eligible psychiatry residents. A current CBT is confirmed; the official sources reviewed did not establish a current OSCE requirement.

How many questions are on the official national CBT examination?

The official item count and duration were not published in the official sources reviewed. A May 2026 national CBT sitting is confirmed.

What standard-setting method determines the passing score?

The current passing threshold and standard-setting method were not published in the official sources reviewed; confirm with Kolegium Psikiatri Indonesia.

What is the scope of content covered on the exam?

The examination scope is defined by Perkonsil No. 104 Tahun 2021 (Standar Pendidikan Profesi Dokter Spesialis Kedokteran Jiwa), covering clinical diagnostic classifications (PPDGJ-III, ICD-10, DSM-5), adult psychopathology, psychopharmacology, child and adolescent psychiatry, geriatric psychiatry, addiction psychiatry, consultation-liaison psychiatry, forensic psychiatry, psychotherapy, and psychiatric emergencies.

Why is this practice bank presented in English?

This practice bank is an independent English-language four-option MCQ study adaptation developed by OpenExamPrep for psychiatric concepts, diagnostic reasoning, and evidence-based psychopharmacology review while preserving exact clinical nomenclature. The official assessment language was not published in the primary sources reviewed.