Free Practice Questions for UKOM Sp.DVE

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Exam Review

Key Facts: UKOM Sp.DVE Exam

Not published

Official written-exam item count

Kolegium DVE National Exam Format

Not published

Written-exam duration

Kolegium DVE

Not published

OSCE station count

Kolegium DVE OSCE Guidelines

Not published

National first-attempt pass rate

Indonesian University PPDS Data

Perkonsil 44/2016

National Competency Standard

Konsil Kedokteran Indonesia

Level 4

Exit Competency Standard

KKI Specialist Medical Competency Framework

The UKOM Sp.DVE is Indonesia's current written-and-OSCE specialist competency examination. This OpenExamPrep bank provides an independent 100-question English-language MCQ study adaptation across published specialty competencies; it does not reproduce official items or simulate OSCE performance.

Sample UKOM Sp.DVE Practice Questions

Try these sample questions to review concepts for the UKOM Sp.DVE exam. Each question includes a detailed explanation. Start the interactive quiz above for the full 100+ question experience with AI tutoring.

1A 4-year-old child presents with widespread, fragile, flaccid bullae and superficial erosions with thin golden-yellow crusts on the perioral and diaper areas. Nikolsky sign is positive on perilesional skin, but the oral mucosa is completely spared. A bacterial culture from intact bulla fluid grows Staphylococcus aureus. Which virulence factor is specifically responsible for the intraepidermal cleavage observed in this patient?

A.Exfoliative toxin A targeting desmoglein 1 within the stratum granulosum

B.Alpha-toxin causing pore formation in keratinocyte cell membranes

C.Toxic shock syndrome toxin-1 acting as a systemic T-cell superantigen

D.Pantone-Valentine leukocidin inducing dermal leukocyte necrosis

Explanation: Bullous impetigo and staphylococcal scalded skin syndrome (SSSS) are mediated by Staphylococcal exfoliative toxins (ETA and ETB), which act as serine proteases that specifically cleave desmoglein 1 (Dsg1). Because Dsg1 is concentrated in the superficial epidermis, this cleavage produces blister formation exclusively within the subcorneal or granular layer, sparing mucous membranes where desmoglein 3 predominates.

2A 48-year-old man undergoing intensive chemotherapy for acute myeloid leukemia develops profound neutropenia (absolute neutrophil count 150/mcL) and high-grade fever. Physical examination reveals an isolated indurated, erythematous macule on the right medial thigh that rapidly evolves over 18 hours into a hemorrhagic bulla surrounded by a violaceous halo, subsequently sloughing into a central black necrotic eschar. What is the underlying pathophysiological mechanism of this cutaneous manifestation?

A.Bacterial invasion of dermal media and adventitia with secondary vascular thrombosis and ischemic necrosis

B.Immune-complex deposition within dermal venules triggering leukocytoclastic necrotizing vasculitis

C.Disseminated intravascular coagulation resulting in generalized dermal microthrombosis without localized bacteria

D.Direct mechanical occlusion of small dermal capillaries by dense clumps of malignant myeloblasts

Explanation: Ecthyma gangrenosum is a classic cutaneous manifestation of Pseudomonas aeruginosa bacteremia in immunocompromised, neutropenic patients. Pseudomonas directly invades the adventitia and media of dermal and subcutaneous blood vessels, producing necrotizing septic vasculitis, vascular thrombosis, and subsequent ischemic infarction with central black eschar formation.

3A 56-year-old woman presents with acute onset of high fever, chills, and a tender, bright red, shiny plaque on her left cheek with well-demarcated, sharply elevated borders that clearly differentiate it from uninvolved skin. The lesion extends across the bridge of the nose. Which clinical feature best differentiates this condition (erysipelas) from non-purulent cellulitis?

A.Involvement of superficial cutaneous lymphatics producing distinct raised margins

B.Presence of purulent subcutaneous fluctuance requiring surgical decompression

C.Primary localization within the deep fascial planes beneath the reticular dermis

D.Obligate etiology due to methicillin-resistant Staphylococcus aureus

Explanation: Erysipelas is a superficial dermal infection involving upper dermis and cutaneous superficial lymphatics, characteristically caused by Streptococcus pyogenes (Group A Streptococcus). The hallmark that differentiates erysipelas from cellulitis is its bright erythematous appearance with sharply demarcated, distinctly elevated borders, whereas cellulitis involves deeper dermis and subcutaneous tissues with indistinct, flat margins.

4A 35-year-old male with poorly controlled type 2 diabetes presents with an extremely painful, confluent inflammatory conglomerate of several interconnected draining nodules on his posterior neck. Thick purulent discharge exudes from multiple follicular openings. What is the most appropriate primary clinical management strategy?

A.Incision and drainage of fluctuant areas combined with systemic anti-staphylococcal antibiotics

B.Topical mupirocin 2% ointment monotherapy applied three times daily for two weeks

C.Immediate wide margin surgical excision with primary closure under local anesthesia

D.High-potency topical corticosteroid application to reduce dermal follicular inflammation

Explanation: The patient has a carbuncle, which is an extensive suppurative infection formed by a coalescence of multiple adjacent furuncles draining through multiple follicular apertures into the deep subcutaneous tissue, commonly caused by Staphylococcus aureus. Primary management requires incision and drainage of fluctuant cavities along with systemic anti-staphylococcal antimicrobial coverage, particularly in diabetic or immunocompromised individuals.

5A 45-year-old obese man presents with well-demarcated, reddish-brown, slightly scaly macules in both axillae and groins that have been asymptomatic for six months. Wood's lamp examination of the lesions reveals a striking, brilliant coral-red fluorescence. What is the causative pathogen of this condition?

A.Corynebacterium minutissimum

B.Malassezia furfur

C.Pseudomonas aeruginosa

D.Candida albicans

Explanation: Erythrasma is a superficial cutaneous infection caused by Corynebacterium minutissimum, a Gram-positive coryneform bacillus. Under Wood's lamp (365 nm), the lesions characteristically emit brilliant coral-red fluorescence due to the bacterial production and cutaneous accumulation of coproporphyrin III.

6A 28-year-old woman presents with recurrent, painful, grouped vesicles on an erythematous base along the vermilion border of her lower lip. A bed-side Tzanck smear is performed on fluid obtained from a freshly unroofed vesicle. Which microscopic finding confirms a herpesvirus infection?

A.Multinucleated giant cells with nuclear molding and ballooning degeneration

B.Large intracytoplasmic molluscum bodies inside enlarged keratinocytes

C.Septate branching hyphae with arthroconidia invading keratinized squames

D.Acantholytic cells with intact, round vesicular nuclei in suprabasal clefts

Explanation: A Tzanck smear of an active Herpes Simplex Virus (HSV) or Varicella Zoster Virus (VZV) vesicle demonstrates characteristic multinucleated epithelial giant cells, nuclear molding, chromatin margination, and keratinocyte ballooning degeneration. While Tzanck smear cannot distinguish between HSV-1, HSV-2, and VZV, it rapidly confirms a herpesvirus family infection.

7A 64-year-old man presents with painful, unilateral vesicular lesions on an erythematous base strictly distributed along his right forehead and upper eyelid. Vesicles are also noted at the tip and side of his nose (Hutchinson's sign). What is the critical clinical implication of this physical finding?

A.Involvement of the nasociliary branch of the ophthalmic nerve (V1), indicating high risk for ocular complications

B.Spread along the infraorbital nerve branch of the maxillary division (V2), predicting intractable facial neuralgia

C.Primary involvement of the facial nerve (cranial nerve VII), indicating impending peripheral Bell's palsy

D.Dissemination to the cervical sympathetic ganglion, predisposing to Horner's syndrome

Explanation: Hutchinson's sign in herpes zoster refers to vesicles on the tip, side, or ala of the nose, which indicates involvement of the external nasal branch of the nasociliary nerve (a branch of the ophthalmic division of the trigeminal nerve, V1). Because the nasociliary nerve also innervates the cornea, iris, and ciliary body, its presence strongly correlates with ocular complications (keratitis, anterior uveitis, secondary glaucoma), warranting immediate ophthalmology consultation.

8A 70-year-old woman experiences severe, lancinating, and burning pain across her right thoracic dermatome (T5-T6) that persists four months after complete re-epithelialization of her herpes zoster vesicular rash. Light brushing of a cotton swab across the skin triggers excruciating pain (allodynia). Which medication is considered a first-line oral systemic agent for this condition?

A.Gabapentin or pregabalin

B.Oral valacyclovir 1000 mg three times daily

C.Prednisone 40 mg daily taper over two weeks

D.Carbamazepine 400 mg twice daily

Explanation: The patient has post-herpetic neuralgia (PHN), defined as neuropathic dermatomal pain persisting beyond 90 days after the onset of herpes zoster rash. First-line pharmacotherapies include alpha-2-delta calcium channel ligands (gabapentin or pregabalin), tricyclic antidepressants (amitriptyline, nortriptyline), and topical 5% lidocaine patches. Antivirals and systemic corticosteroids do not treat established chronic neuropathic pain.

9A 6-year-old boy presents with multiple discrete, asymptomatic, dome-shaped, pearl-like papules with central dells (umbilication) on the trunk and axillae. His mother notes that expressing a lesion produces a small curdy white plug. A punch biopsy reveals large, oval, homogeneous, intracytoplasmic inclusion bodies within the stratum spinosum that displace the host keratinocyte nucleus. What is this histological finding called?

A.Henderson-Paterson bodies

B.Cowdry A inclusion bodies

C.Guarnieri bodies

D.Negri bodies

Explanation: Henderson-Paterson bodies (molluscum bodies) are large, eosinophilic to basophilic intracytoplasmic inclusion bodies pathognomonic for molluscum contagiosum (caused by a double-stranded DNA Molluscipoxvirus). They represent virion-packed structures within keratinocytes that expand from the stratum basale to the stratum corneum, eventually shedding from the central umbilication.

10A 22-year-old man presents with multiple rough, hyperkeratotic, exophytic papules on the dorsal hands and periungual regions. Dermoscopy reveals multiple tightly packed papillae with central red or black dots representing thrombosed capillaries. Histopathologic examination shows hyperkeratosis, papillomatosis, and cells in the upper spinous and granular layers exhibiting pyknotic eccentric nuclei surrounded by a clear cytoplasmic halo. What is the specific name for these altered keratinocytes?

A.Koilocytes

B.Civatte bodies

C.Corp ronds

D.Grains

Explanation: Koilocytes are keratinocytes altered by human papillomavirus (HPV) infection (in verruca vulgaris, verruca plana, or condylomata acuminata). Histologically, koilocytes demonstrate nuclear enlargement, irregular hyperchromatic pyknotic nuclei, and a prominent perinuclear clear halo with coarsened keratohyalin granules.

About the UKOM Sp.DVE Exam

The UKOM Sp.DVE is the national competency examination for eligible dermatology, venereology, and aesthetics specialist residents in Indonesia. Kolegium DVE and Konsil Kesehatan Indonesia conducted a current written-and-OSCE Ukomnas in May 2026; Perkonsil No. 44 Tahun 2016 supplies published education and competency scope.

Exam sponsor: Kolegium DVE / KKI. The requirements and fees below concern the certification or admission exam, separate from our free practice resources.

Assessment

Question count not published by the exam provider

Time Limit

Not published in the official sources reviewed.

Passing Score

Not published in the official sources reviewed.

Exam / Certification Fees

Not published in the official sources reviewed.

Exam sponsor website

Reported exam pass rate: Not published in the official sources reviewed.. Not published in the official sources reviewed. Exam sponsor website

Fees, eligibility, and exam policies can change. Confirm them with the exam sponsor before applying or paying.

Official sources

Our practice resources: topics covered

We aim to reflect publicly available exam outlines and topic information in our study resources. Coverage, format, and difficulty may differ from the actual exam, and we cannot guarantee that every detail is accurate or current. Confirm exam requirements, fees, and policies with the official exam sponsor.

25%

Infectious Dermatoses & Tropical Dermatology

Bacterial infections (impetigo, erysipelas, ecthyma gangrenosum); viral exanthems (HSV, VZV, HPV, molluscum); fungal mycoses (dermatophytoses, chromoblastomycosis, sporotrichosis); parasitic infestations (scabies, larva migrans); Morbus Hansen / Leprosy (Ridley-Jopling classification, slit-skin smear BI/MI, WHO MDT regimens, Type 1 reversal and Type 2 ENL reactions, neuritis); cutaneous tuberculosis (scrofuloderma, lupus vulgaris).

25%

Inflammatory, Papulosquamous & Autoimmune Bullous Dermatoses

Atopic dermatitis (Hanifin-Rajka criteria, filaggrin, dupilumab); contact dermatitis (patch testing); seborrheic dermatitis; stasis dermatitis; psoriasis vulgaris (Auspitz sign, pustular von Zumbusch, methotrexate, anti-IL-17/IL-23 biologics); pityriasis rosea; lichen planus; autoimmune bullous disorders (pemphigus vulgaris vs foliaceus, bullous pemphigoid, dermatitis herpetiformis, linear IgA); cutaneous lupus erythematosus; dermatomyositis.

20%

Sexually Transmitted Infections (Venereology)

Syphilis (primary chancre, secondary condylomata lata, latent syphilis staging, neurosyphilis, non-treponemal vs treponemal testing, benzathine penicillin G regimens, Jarisch-Herxheimer reaction); gonococcal and non-gonococcal urethritis; genital ulcer diseases (HSV, chancroid, LGV, donovanosis); anogenital warts; pelvic inflammatory disease; bacterial vaginosis; trichomoniasis; syndromic STI management.

15%

Severe Cutaneous Adverse Drug Reactions (SCAR) & Cutaneous Oncology

Severe adverse drug reactions (SJS/TEN, SCORTEN prognostic score, HLA-B*15:02 and HLA-B*58:01 pharmacogenomics, DRESS/DIHS with HHV-6 reactivation, AGEP); benign skin tumors (seborrheic keratosis, dermatofibroma); premalignant actinic keratosis; basal cell carcinoma (nodular, infiltrative, Mohs surgery); squamous cell carcinoma (Marjolin ulcer); cutaneous melanoma (acral lentiginous, Breslow thickness, excision margins).

15%

Aesthetic Dermatology, Laser & Dermatologic Surgery

Acne vulgaris (pathogenesis, microcomedo, benzoyl peroxide, oral isotretinoin cumulative dosing); rosacea subtypes; pigmentary disorders (melasma Kligman formula, vitiligo OMP steroids and phototherapy); alopecia areata and telogen effluvium; chemical peels (salicylic acid, TCA CROSS); botulinum toxin complications (ptosis management); dermal filler vascular emergencies and hyaluronidase protocol; selective photothermolysis, Nd:YAG and fractional lasers in dark skin; skin biopsy techniques; local anesthesia.

Preparing for the UKOM Sp.DVE Exam

What You Need to Know

Passing score: Not published in the official sources reviewed.
Assessment: Question count not published by the exam provider
Time limit: Not published in the official sources reviewed.
Exam / certification fees: Not published in the official sources reviewed. Official sources

Using Our Practice Resources

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UKOM Sp.DVE: Suggested Study Strategy

1Master Morbus Hansen classifications, WHO-MDT blister pack components, and reaction management: distinguish Type 1 reversal reaction (delayed hypersensitivity, neuritis, high-dose oral prednisone) from Type 2 ENL (immune-complex vasculitis, systemic fever, thalidomide and clofazimine).

2Memorize immunobullous direct immunofluorescence patterns: intercellular 'fishnet' IgG/C3 with anti-Dsg3/Dsg1 in pemphigus vulgaris vs linear basement membrane zone IgG/C3 with anti-BP180/BP230 in bullous pemphigoid vs granular IgA in dermal papillae in dermatitis herpetiformis.

3Review the SCORTEN prognostic scoring criteria for SJS/TEN: memorize all 7 parameters and know the HLA-B*15:02 screening indication for carbamazepine in Southeast Asian and Indonesian patients.

4Drill STI treatment regimens: single-dose IM ceftriaxone 500 mg for gonorrhea, oral doxycycline 100 mg BID x 7 days for chlamydia, benzathine penicillin G dosing by syphilis stage, and high-dose hyaluronidase emergency protocols for filler vascular occlusion.

5Understand dermatologic laser physics and Fitzpatrick skin types IV-V: master selective photothermolysis, long-pulsed Nd:YAG 1064 nm safety for pigment and hair removal in darker skin phototypes, and PIH prevention.

Frequently Asked Questions

What is the difference between Sp.KK and Sp.DVE in Indonesia?

In accordance with updated medical council regulations and professional nomenclatures established by the Konsil Kesehatan Indonesia (KKI) and Kolegium DVE, the traditional specialist title Sp.KK (Spesialis Kulit dan Kelamin) was formally transitioned to Sp.DVE (Spesialis Dermatologi, Venereologi, dan Estetika). This reflects the comprehensive scope of modern specialty practice spanning clinical dermatology, venereology / sexually transmitted infections, and aesthetic dermatology.

What is the format of the official UKOM Sp.DVE exit examination?

Current sources confirm a written examination and an Objective Structured Clinical Examination. Item count, duration and station count were not published in the official sources reviewed.

Who is eligible to take the UKOM Sp.DVE examination?

Candidates must be medical doctors enrolled in an accredited university PPDS (Program Pendidikan Dokter Spesialis) program in Dermatology, Venereology, and Aesthetics in Indonesia who have completed all required clinical semesters, satisfied all logbook requirements under Perkonsil No. 44 Tahun 2016, and received formal written endorsement from their Residency Program Director (KPS).

How often is the national UKOM Sp.DVE examination held?

The national examination is typically administered twice a year, usually in May and November, coordinated by the Tim Ukomnas Kolegium DVE and KKI across designated regional academic medical centers in Indonesia.

Is this OpenExamPrep question bank an official examination?

No. This practice question bank is an independent English-language MCQ study adaptation developed by OpenExamPrep for self-directed clinical revision and board preparation. It is not affiliated with, endorsed by, or sponsored by Kolegium DVE, PERDOSKI, or KKI.

Why are tropical diseases like Morbus Hansen emphasized in the exam?

Indonesia remains one of the highest leprosy (Morbus Hansen) burden countries globally. Consequently, the national curriculum established by Perkonsil No. 44/2016 mandates high-level expert competency (Level 4 competency) in the diagnosis, slit-skin smear evaluation, WHO-MDT regimens, early detection of nerve function impairment, and management of Type 1 and Type 2 leprosy reactions.