100+ Free TEP Patologia Practice Questions

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Practice Questions100 questions

2026 Statistics

Key Facts: TEP Patologia Exam

50 Items

Multiple-choice questions on the official TEP Prova Teórica

Edital TEP — SBP / AMB

3 Stages

Evaluation components (Theoretical MCQ, Digital Microscopy, and Macroscopy/Autopsy)

Edital TEP — SBP / AMB

R$ 950,00

Registration fee for SBP + AMB fully affiliated members (R$ 3.000 for non-members)

Edital SBP / AMB

7.0 / 10.0

Minimum final weighted average grade required for passing

Regulamento Oficial SBP / AMB

RQE Patologia

Specialist Registration Credential Conferred with CFM (Anatomia Patológica)

Conselho Federal de Medicina (CFM) / AMB

Annual

Official Examination Frequency Conducted by SBP / AMB

Sociedade Brasileira de Patologia (SBP)

The TEP (Título de Especialista em Patologia) is the premier Brazilian pathology board certification examination administered annually by SBP and AMB. It combines theoretical testing on disease mechanisms, WHO tumor classifications, and molecular biomarkers with practical evaluations of digital histopathology, cytopathology, gross pathology, and autopsy sequencing.

Sample TEP Patologia Practice Questions

Try these sample questions to test your TEP Patologia exam readiness. Each question includes a detailed explanation. Start the interactive quiz above for the full 100+ question experience with AI tutoring.

1During a routine microscopic review of a myocardial biopsy from a patient who suffered an acute myocardial infarction 24 hours prior, the pathologist observes preservation of the basic structural outline of necrotic cardiomyocytes with loss of nuclei (karyolysis) and intense cytoplasmic eosinophilia, accompanied by an early neutrophilic infiltrate. What type of necrosis is depicted by these morphological findings?

A.Coagulative necrosis

B.Liquefactive necrosis

C.Caseous necrosis

D.Fibrinoid necrosis

Explanation: Coagulative necrosis is the characteristic pattern of cell death in hypoxic/ischemic infarction in all solid organs except the brain. It is characterized by denaturation of structural proteins and enzymes, which blocks proteolysis and preserves the microscopic architecture and 'tombstone' outlines of dead cells for several days.

2A researcher is studying apoptosis pathways in lymphoid cells exposed to DNA-damaging radiation. The signaling cascade involves cellular stress, activation of pro-apoptotic BH3-only proteins (such as BIM, BID, and PUMA), neutralization of anti-apoptotic BCL-2/BCL-XL, permeabilization of the outer mitochondrial membrane via BAX and BAK oligomerization, and cytoplasmic release of cytochrome c. Which initiator caspase is directly activated by the downstream formation of the apoptosome complex (cytochrome c + APAF-1)?

A.Caspase-8

B.Caspase-9

C.Caspase-10

D.Caspase-1

Explanation: In the intrinsic (mitochondrial) pathway of apoptosis, release of cytochrome c into the cytoplasm induces binding to apoptotic protease activating factor-1 (APAF-1) and procaspase-9 to form the multimeric apoptosome complex. This results in the autocatalytic activation of initiator caspase-9, which subsequently cleaves and activates executioner caspases (caspase-3 and caspase-7).

3An endoscopic biopsy of the distal esophagus in a 55-year-old patient with long-standing gastroesophageal reflux disease reveals replacement of the normal non-keratinized stratified squamous epithelium by simple columnar epithelium containing distinct goblet cells (intestinal metaplasia). How is this adaptive cellular process best classified?

A.Hypertrophy

B.Dysplasia

C.Metaplasia

D.Anaplasia

Explanation: Metaplasia is a reversible adaptive change in which one differentiated adult cell type (in this case, esophageal squamous epithelium) is replaced by another adult cell type (columnar epithelium with goblet cells, characteristic of Barrett esophagus) better suited to survive hostile environmental conditions such as chronic gastric acid and bile exposure.

4During the vascular and cellular response of acute inflammation, circulating leukocytes exit the microvasculature to reach the site of injury. Which family of endothelial adhesion molecules is primarily responsible for the initial loose attachment and rolling of neutrophils along the activated endothelium?

A.Integrins (such as LFA-1 and Mac-1)

B.Immunoglobulin superfamily molecules (ICAM-1 and VCAM-1)

C.Cadherins (E-cadherin and VE-cadherin)

D.Selectins (E-selectin and P-selectin)

Explanation: Leukocyte rolling is mediated by selectins (P-selectin and E-selectin on endothelial cells, and L-selectin on leukocytes) binding to sialylated oligosaccharide ligands (such as Sialyl-Lewis X). This weak and transient interaction allows leukocytes to roll slowly along the vessel wall before high-affinity integrin-mediated firm adhesion occurs.

5Histopathological examination of a lung nodule resected from a 42-year-old patient reveals well-formed granulomas composed of aggregated epithelioid histiocytes, Langhans multinucleated giant cells with horseshoe-arranged peripheral nuclei, and central acellular granular caseous necrosis. Which cytokine produced by CD4+ T-helper 1 (Th1) cells is the principal mediator driving the transformation of recruited macrophages into epithelioid cells and giant cells in this setting?

A.Interferon-gamma (IFN-γ)

B.Interleukin-4 (IL-4)

C.Interleukin-10 (IL-10)

D.Transforming Growth Factor-beta (TGF-β)

Explanation: Interferon-gamma (IFN-γ) secreted by antigen-activated Th1 lymphocytes is the master activating cytokine of the classical macrophage activation pathway (M1). It stimulates macrophages to increase microbicidal activity, upregulate lysosomal enzymes and nitric oxide synthase, and transform morphologically into aggregated epithelioid histiocytes and multinucleated Langhans giant cells in immune granulomas.

6During cutaneous wound healing and tissue repair, fibroblasts transform into contractile myofibroblasts to facilitate wound contraction and extracellular matrix remodeling. Which growth factor is recognized as the most potent and critical driver of myofibroblast differentiation, fibroblast proliferation, and type I/III collagen synthesis in tissue fibrosis?

A.Epidermal Growth Factor (EGF)

B.Transforming Growth Factor-beta (TGF-β)

C.Vascular Endothelial Growth Factor A (VEGF-A)

D.Fibroblast Growth Factor 2 (FGF-2)

Explanation: Transforming Growth Factor-beta (TGF-β) is the master cytokine driving tissue fibrosis and scarring. It promotes the transdifferentiation of fibroblasts into alpha-smooth muscle actin (α-SMA)-positive myofibroblasts, stimulates the synthesis of collagen and fibronectin, and suppresses matrix metalloproteinases (MMPs) to reduce extracellular matrix degradation.

7At autopsy of a 68-year-old individual who died suddenly, a large occlusive intravascular mass is identified in the abdominal aorta overlying an ulcerated atheromatous plaque. Gross and microscopic inspection reveals distinct alternating pale laminar bands of platelets and fibrin admixed with darker layers rich in red blood cells (Lines of Zahn). What is the primary diagnostic significance of Lines of Zahn?

A.They signify a postmortem 'currant-jelly' blood clot formed by gravitational sedimentation.

B.They confirm the presence of fat embolism from long-bone fractures.

C.They prove that the thrombus formed in a living individual under active blood flow (antemortem thrombosis).

D.They are pathognomonic for amniotic fluid embolism in maternal vessels.

Explanation: Lines of Zahn are microscopic and gross laminations consisting of pale alternating layers of platelets and fibrin interspersed with darker erythrocyte-rich layers. They represent the hallmark of antemortem thrombosis formed under pulsatile or active laminar blood flow, distinguishing true thrombi from postmortem jelly clots (which lack structural laminations and are unattached to vascular walls).

8A 28-year-old primigravida experiences sudden collapse, severe dyspnea, cyanosis, hypotension, and seizures followed by disseminated intravascular coagulation (DIC) during labor. Histological sections of the maternal pulmonary microvasculature at autopsy reveal impacted fetal squamous squames, mucin droplets, and lanugo hairs. Which form of embolism is responsible for this catastrophic obstetric complication?

A.Fat embolism syndrome

B.Air embolism

C.Paradoxical thromboembolism

D.Amniotic fluid embolism

Explanation: Amniotic fluid embolism occurs when tears in the placental membranes or uterine veins allow amniotic fluid containing fetal debris (anucleate squamous squames, lanugo hairs, vernix caseosa, and meconium mucin) to enter the maternal venous circulation. The entry of procoagulant thromboplastic substances triggers massive pulmonary vasoconstriction, cardiogenic shock, and severe consumptive coagulopathy (DIC).

9An autopsy performed on a patient who succumbed to severe septic shock secondary to peritonitis demonstrates lungs that are heavy, boggy, and dark red. Histopathologic sections show diffuse alveolar damage with dense, eosinophilic hyaline membranes lining dilated alveolar ducts, extensive alveolar edema, and microvascular congestion. What is the clinicopathological diagnosis for this pulmonary response to systemic shock?

A.Acute Respiratory Distress Syndrome (ARDS) / Diffuse Alveolar Damage

B.Usual Interstitial Pneumonia (UIP)

C.Pulmonary Alveolar Proteinosis

D.Desquamative Interstitial Pneumonia (DIP)

Explanation: Diffuse Alveolar Damage (DAD) is the histologic correlate of Acute Respiratory Distress Syndrome (ARDS). In the acute exudative phase (days 1 to 7), endothelial and type I alveolar pneumocyte injury leads to capillary leakage, interstitial edema, and formation of prominent eosinophilic hyaline membranes composed of fibrin, plasma proteins, and necrotic cell debris lining alveolar ducts.

10Malignant neoplastic cells frequently reprogram their cellular metabolism to favor aerobic glycolysis—metabolizing glucose into lactate even in the presence of abundant oxygen—to generate biosynthetic intermediates for rapid growth and proliferation. What is the classic eponym used in tumor biology to designate this metabolic hallmark of cancer?

A.Pasteur effect

B.Warburg effect

C.Crabtree effect

D.Cori cycle

Explanation: The Warburg effect (aerobic glycolysis) is a hallmark of cancer described by Otto Warburg, wherein tumor cells exhibit high rates of glucose uptake and conversion to lactate despite ample oxygen. This pathway provides carbon skeletons and nucleotide precursors necessary for rapid cell division and is the metabolic basis for 18F-FDG PET imaging in oncology.

About the TEP Patologia Exam

The Título de Especialista em Patologia (TEP) is the official board certification for medical pathologists in Brazil, awarded by the Sociedade Brasileira de Patologia (SBP) in partnership with the Associação Médica Brasileira (AMB) and registered with the Conselho Federal de Medicina (CFM). The examination evaluates comprehensive proficiency in modern surgical pathology, histomorphology, diagnostic immunohistochemistry, molecular pathology, cytopathology reporting terminologies, and autopsy pathology. Achieving the TEP title is the definitive requirement for Brazilian pathologists to obtain their Registro de Qualificação de Especialista (RQE) in Pathology (Anatomia Patológica).

Assessment

Tripartite examination conducted in-person: (1) Prova Teórica: 50 multiple-choice questions evaluating general pathology, systemic surgical pathology, cytopathology, immunohistochemistry, and molecular pathology; (2) Prova de Microscopia: Digital whole-slide image evaluation assessing diagnostic accuracy on biopsies, resections, and cytology smears; (3) Prova de Macroscopia e Autópsia: Macroscopic specimen description, surgical margins, TNM staging, autopsy clinicopathological correlation, and perinatal pathology.

Time Limit

Full-day multi-stage examination sessions as designated by the annual SBP/AMB Edital

Passing Score

A mean of at least 7.0 across the three papers; alternatively, a candidate reaching 7.0 in only one or two papers is approved provided the final weighted mean [(teórica × 4 + microscopia × 4 + macroscopia/autópsia × 2) ÷ 10] is at least 7.0

Exam Fee

R$ 950,00 (sócios adimplentes SBP + AMB) / R$ 1.000,00 (sócios adimplentes SBP) / R$ 3.000,00 (não sócios) (Sociedade Brasileira de Patologia (SBP) — Associação Médica Brasileira (AMB))

TEP Patologia Exam Content Outline

15%

Patologia Geral e Mecanismos Moleculares de Doença

Cell injury, necrosis types (coagulative, liquefactive, caseous, fat, fibrinoid), apoptosis pathways (intrinsic/extrinsic), acute/chronic inflammation mediators, tissue repair and fibrosis, hemodynamic alterations (edema, thrombosis, embolism, shock), hallmarks of cancer, oncogenes, tumor suppressor pathways (TP53, RB1, APC, BRCA1/2), epigenetics, and tumor grading/staging principles.

15%

Patologia Gastrointestinal e Hepatobiliopancreática

Esophageal diseases (Barrett esophagus, dysplasia grading, squamous cell carcinoma, adenocarcinoma), gastritis (OLGA/OLGIM staging, H. pylori, autoimmune metaplastic gastritis), gastric cancer (Lauren classification, WHO, HER2, MMR), IBD (Crohn vs. UC) vs. infectious colitis, colorectal polyps (tubular/villous adenomas, sessile serrated lesions, TSA, MSI-H pathway), GIST (c-KIT, PDGFRA, DOG1, risk stratification), viral and metabolic liver diseases (MASH scoring), cirrhosis, hepatic adenoma subtypes, HCC, cholangiocarcinoma, and pancreatic ductal adenocarcinoma vs. neuroendocrine tumors.

15%

Patologia Mamária e Ginecológica

Breast pathology: DCIS architecture and nuclear grade, Nottingham histologic grading (tubule formation, nuclear pleomorphism, mitoses), invasive carcinomas (NST, lobular with CDH1/E-cadherin loss, tubular, mucinous, metaplastic), ASCO/CAP biomarker guidelines (ER, PR, HER2 IHC/FISH reflex algorithm, Ki-67), and fibroepithelial lesions (fibroadenoma vs. phyllodes tumor grading). Gynecologic pathology: Endometrial hyperplasia (EIN criteria), endometrial carcinoma (endometrioid, serous p53 abnormal, clear cell, TCGA/ProMisE molecular classification), cervical intraepithelial lesions (LSIL/HSIL, p16/Ki-67), ovarian neoplasms (high-grade serous, low-grade serous, clear cell, endometrioid, mucinous, granulosa cell tumors), and gestational trophoblastic disease (complete vs. partial hydatidiform mole p57 IHC).

12%

Patologia Geniturinária e Renal

Prostate: Modified Gleason grading and WHO/ISUP Grade Groups (1 through 5), intraductal carcinoma of the prostate, high-grade PIN, neuroendocrine differentiation. Urinary bladder: Urothelial neoplasms (WHO classification: PUNLMP, low-grade and high-grade non-invasive papillary urothelial carcinoma, invasive urothelial carcinoma, divergent differentiation). Kidney: Renal cell carcinoma subtypes (clear cell RCC VHL/3p, papillary RCC MET/PRCC, chromophobe RCC Hale colloidal iron, oncocytoma, TFE3/TFEB translocation RCC, collecting duct carcinoma), WHO/ISUP nuclear grading. Testis: Germ cell tumors (seminoma vs. non-seminomatous GCT, GCNIS, OCT3/4, SALL4, CD30, AFP, hCG).

10%

Patologia Torácica e Pulmonar

Lung adenocarcinoma WHO 2021 subtyping (lepidic, acinar, papillary, micropapillary, solid), adenocarcinoma in situ (AIS) and minimally invasive adenocarcinoma (MIA), squamous cell carcinoma (p40, p63, CK5/6), neuroendocrine tumors (typical carcinoid, atypical carcinoid, small cell lung carcinoma, LCNEC), malignant pleural mesothelioma (epithelioid, sarcomatoid, biphasic; BAP1 loss, MTAP loss, WT1, calretinin), and non-neoplastic pulmonary pathology (idiopathic pulmonary fibrosis / UIP pattern vs. NSIP).

10%

Dermatopatologia, Partes Moles e Patologia Endócrina

Skin: Melanocytic lesions (dysplastic nevus, melanoma in situ, invasive melanoma, Breslow thickness, ulceration, Clark levels, SOX10, Melan-A, PRAME), non-melanoma skin cancer (basal cell carcinoma subtypes, squamous cell carcinoma). Soft tissue: FNCLCC grading of sarcomas (differentiation, mitoses, necrosis), well-differentiated/dedifferentiated liposarcoma (MDM2/CDK4), synovial sarcoma (SS18::SSX), leiomyosarcoma, rhabdomyosarcoma (PAX3/7::FOXO1). Endocrine: Thyroid nodules (Bethesda classification 3rd ed., papillary carcinoma variants and nuclear features, follicular adenoma vs. carcinoma, NIFTP criteria, poorly differentiated and anaplastic carcinoma, medullary carcinoma), adrenal cortical neoplasms (Weiss criteria), and pheochromocytoma (PASS / GAPP scoring).

Hematopatologia e Patologia Linfonodal

Reactive lymphadenopathies (follicular hyperplasia, sinus histiocytosis, necrotizing lymphadenitis / Kikuchi-Fujimoto disease). Mature B-cell neoplasms: Diffuse large B-cell lymphoma (DLBCL, Hans algorithm CD10/BCL6/MUM1 for GCB vs. non-GCB, double-hit / triple-hit lymphomas), follicular lymphoma (grading 1-3A vs. 3B), mantle cell lymphoma (cyclin D1, SOX11), marginal zone lymphoma (MALT), Burkitt lymphoma (MYC, Ki-67 ~100%). Hodgkin lymphoma: Classical Hodgkin lymphoma (nodular sclerosis, mixed cellularity, lymphocyte-rich, lymphocyte-depleted; CD30+, CD15+, PAX5 dim, CD20-, CD45-) vs. Nodular lymphocyte predominant Hodgkin lymphoma (LP cells, CD20+, CD79a+, OCT2+, BOB.1+).

Citopatologia Diagnóstica

Gynecologic cytopathology: The Bethesda System for Reporting Cervical Cytology (NILM, ASC-US, ASC-H, LSIL, HSIL, AGC, adenocarcinoma). Thyroid cytopathology: The Bethesda System for Reporting Thyroid Cytopathology (Categories I to VI, risk of malignancy, management implications). Salivary gland cytopathology: The Milan System for Reporting Salivary Gland Cytopathology. Urinary cytopathology: The Paris System for Reporting Urinary Cytology (criteria for High-Grade Urothelial Carcinoma - HGUC, N/C ratio ≥0.7, hyperchromasia, irregular nuclear membranes).

Imunohistoquímica, Patologia Molecular e Necrópsia / Patologia Forense

Diagnostic immunohistochemical algorithms: Lineage determination (cytokeratin CK7/CK20 coordinates, TTF-1, p40/p63, CDX2, GATA3, SATB2, PAX8, WT1, synaptophysin, chromogranin A, INSM1). Predictive/prognostic biomarkers: Mismatch repair (MMR) proteins (MLH1, MSH2, MSH6, PMS2) and microsatellite instability (MSI), BRAF V600E, EGFR mutations, ALK/ROS1 rearrangements, PD-L1 scoring (TPS, CPS, IC). Autopsy pathology: Gross dissection techniques, postmortem interval changes, death certificate cause-of-death sequence (immediate, intermediate, underlying cause), and perinatal/placental pathology (Amsterdam consensus criteria for maternal/fetal vascular malperfusion, acute chorioamnionitis).

How to Pass the TEP Patologia Exam

What You Need to Know

Passing score: A mean of at least 7.0 across the three papers; alternatively, a candidate reaching 7.0 in only one or two papers is approved provided the final weighted mean [(teórica × 4 + microscopia × 4 + macroscopia/autópsia × 2) ÷ 10] is at least 7.0
Assessment: Tripartite examination conducted in-person: (1) Prova Teórica: 50 multiple-choice questions evaluating general pathology, systemic surgical pathology, cytopathology, immunohistochemistry, and molecular pathology; (2) Prova de Microscopia: Digital whole-slide image evaluation assessing diagnostic accuracy on biopsies, resections, and cytology smears; (3) Prova de Macroscopia e Autópsia: Macroscopic specimen description, surgical margins, TNM staging, autopsy clinicopathological correlation, and perinatal pathology.
Time limit: Full-day multi-stage examination sessions as designated by the annual SBP/AMB Edital
Exam fee: R$ 950,00 (sócios adimplentes SBP + AMB) / R$ 1.000,00 (sócios adimplentes SBP) / R$ 3.000,00 (não sócios)

Keys to Passing

Work through all 100 available questions
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TEP Patologia Study Tips from Top Performers

1Master WHO 5th Edition Classification Blue Books: Focus on updated tumor terminology, essential diagnostic criteria, subtype classification, and defining molecular alterations across organ systems.

2Internalize Immunohistochemistry (IHC) Algorithms: Memorize cytokeratin coordinates (CK7+/CK20- vs. CK7-/CK20+ vs. CK7+/CK20+), lineage-specific markers (TTF-1, p40/p63, CDX2, GATA3, SATB2, PAX8, WT1, Melan-A, SOX10), and neuroendocrine markers (synaptophysin, chromogranin A, INSM1).

3Master Prognostic & Predictive Biomarker Guidelines: Know the exact ASCO/CAP scoring criteria and reflex testing algorithms for HER2 (IHC 0, 1+, 2+ equivocal with FISH reflex, 3+ positive), ER/PR percentage thresholds, Ki-67 indices, and Mismatch Repair (MMR) protein staining patterns (MLH1/PMS2 and MSH2/MSH6 heterodimers).

4Systematize Cytopathology Reporting Systems: Master the diagnostic criteria, terminology, and risk of malignancy (ROM) for The Bethesda System (Cervix and Thyroid 3rd ed.), The Paris System (Urinary), and The Milan System (Salivary Gland).

5Review Tumor Grading & Staging Schemes: Memorize Nottingham histologic grade for breast carcinoma, ISUP/WHO Grade Groups (1–5) for prostate adenocarcinoma, FNCLCC grading for soft tissue sarcomas, and TNM 8th/9th edition staging rules (e.g., depth of invasion in oral cavity, Breslow thickness in melanoma).

6Consolidate Autopsy & Forensic Principles: Understand postmortem interval changes, cause-of-death sequencing on death certificates (Part I line a, b, c vs. Part II contributing causes), and perinatal autopsy protocols (placental pathology Amsterdam consensus).

Frequently Asked Questions

What is the TEP and why is it essential for pathologists in Brazil?

The Título de Especialista em Patologia (TEP) is the official specialist medical board certification awarded by the Sociedade Brasileira de Patologia (SBP) and the Associação Médica Brasileira (AMB). Achieving the TEP allows physicians to register their specialized qualification (Registro de Qualificação de Especialista - RQE) in Pathology / Anatomia Patológica with the Regional Medical Councils (CRMs) and Federal Council of Medicine (CFM), which is legally required to sign surgical pathology, cytopathology, and autopsy reports as a specialist in Brazil.

What are the prerequisite eligibility requirements to sit for the TEP examination?

Candidates must be fully licensed physicians registered with a Regional Medical Council (CRM) in Brazil and fulfill one of the eligible pathways: (1) Completion of an accredited Medical Residency Program (CNRM/MEC) in Pathology (Anatomopatologia); (2) Completion of an SBP-accredited pathology specialization training program; or (3) Proven clinical practice in Anatomical Pathology for at least double the duration of official residency (6 years), substantiated by official institutional documentation and passing curricular score evaluation as detailed in the annual SBP/AMB Edital.

How is the TEP examination structured across its different stages?

The examination is divided into three distinct evaluated sections: (1) Prova Teórica, consisting of 50 multiple-choice questions on general pathology, systemic surgical pathology, cytopathology, immunohistochemistry, and molecular diagnostics; (2) Prova de Microscopia, evaluating digital whole-slide images for diagnostic histopathology and cytopathology; and (3) Prova de Macroscopia e Autópsia, presenting gross pathology photographs, surgical margin assessment, TNM staging, autopsy case sequencing, and perinatal pathology.

What is the passing score and grading criteria for the TEP?

To pass the TEP examination and be awarded the specialist title, candidates must obtain a final weighted composite grade equal to or greater than 7.0 (on a 0.0 to 10.0 scale) across the theoretical, microscopic, and macroscopic/autopsy examination components, adhering to the eliminatory cutoffs specified in the official annual Edital.

Which classifications and international consensus guidelines are tested on the TEP?

The examination tests current international diagnostic standards, including the World Health Organization (WHO) Classification of Tumours (5th Edition Blue Books), CAP/ASCO biomarker guidelines (HER2, ER/PR, Ki-67, MMR, PD-L1), The Bethesda System for Reporting Cervical Cytology, The Bethesda System for Reporting Thyroid Cytopathology (3rd Edition), The Paris System for Reporting Urinary Cytology, The Milan System for Salivary Gland Cytopathology, FNCLCC sarcoma grading, Nottingham breast cancer grading, and the modified Gleason / ISUP Grade Group system for prostate cancer.

Why is this OpenExamPrep practice bank presented in English?

This practice bank is an English-language MCQ study adaptation, not a replica of the official paper: the real TEP examination is sat in Portuguese and incorporates practical digital slide and macroscopy stations. This question bank is designed to support international pathology fellows, Brazilian residents preparing for board examinations who study global literature, and pathologists worldwide, while preserving authentic terminology, IHC panel phenotypes, and diagnostic criteria.