100+ Free TEN Neurologia Practice Questions

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Key Facts: TEN Neurologia Exam

100 Items

Multiple-choice questions across theoretical and practical board evaluation stages

Edital TEN — ABN / AMB

7 Hours

Total examination duration covering theoretical and multimedia/practical sessions

Edital TEN — ABN / AMB

R$ 1.950,00

Registration fee for ABN / AMB affiliated members (R$ 3.900 for non-members)

Edital ABN / AMB

70%

Minimum overall passing score threshold (7.0 / 10.0 scale)

Regulamento Oficial ABN / AMB

RQE Neurologia

Specialist registration credential awarded with CFM

Conselho Federal de Medicina (CFM) / AMB

Annual

Official examination frequency conducted by ABN / AMB

Academia Brasileira de Neurologia (ABN)

The TEN (Título de Especialista em Neurologia) is the national neurology board certification examination administered annually by the ABN and AMB. It consists of theoretical and practical/multimedia stages evaluating diagnostic criteria (ILAE 2017, McDonald 2017, ICHD-3, DAWN/DEFUSE-3), neuroimaging, neurophysiology, and guideline-directed neurological therapeutics.

Sample TEN Neurologia Practice Questions

Try these sample questions to test your TEN Neurologia exam readiness. Each question includes a detailed explanation. Start the interactive quiz above for the full 100+ question experience with AI tutoring.

1A 62-year-old man presents to the emergency department 75 minutes after the sudden onset of right hemiparesis and global aphasia. His baseline modified Rankin Scale (mRS) is 0. Non-contrast cranial computed tomography (CT) reveals an ASPECTS score of 9 with no acute hemorrhage or early ischemic changes. His blood pressure is 172/96 mmHg, blood glucose is 134 mg/dL, and international normalized ratio (INR) is 1.0. According to current Brazilian and international acute ischemic stroke guidelines, what is the most appropriate immediate medical intervention?

A.Initiate intravenous thrombolysis with recombinant tissue plasminogen activator (Alteplase 0.9 mg/kg or Tenecteplase 0.25 mg/kg) without waiting for further imaging.

B.Administer oral aspirin 300 mg immediately and order urgent brain magnetic resonance imaging (MRI) with diffusion-weighted imaging (DWI).

C.Reduce blood pressure with intravenous nitroprusside until systolic blood pressure is below 130 mmHg before considering thrombolytic therapy.

D.Initiate therapeutic intravenous unfractionated heparin with a targeted activated partial thromboplastin time (aPTT) of 1.5–2.5 times control.

Explanation: Intravenous thrombolysis (IV rtPA / Alteplase 0.9 mg/kg or Tenecteplase 0.25 mg/kg) is recommended for eligible acute ischemic stroke patients presenting within 4.5 hours of symptom onset once intracranial hemorrhage is excluded by non-contrast CT. Blood pressure is already below the guideline-specified threshold for thrombolysis (<185/110 mmHg), allowing immediate treatment without delay.

2A 68-year-old woman was last seen well at 10:00 PM when going to bed. At 7:00 AM the next morning (9 hours later), her family finds her with dense left-sided hemiplegia and neglect (NIHSS score of 17). Non-contrast cranial CT reveals no intracranial hemorrhage and an ASPECTS score of 8. CT angiography confirms an occlusion of the M1 segment of the right middle cerebral artery. Perfusion CT demonstrates an ischemic core volume of 18 mL and a total hypoperfused penumbral volume of 94 mL (mismatch volume of 76 mL; mismatch ratio > 5.0). Based on the DAWN and DEFUSE-3 clinical trial criteria, what is the indicated management?

A.Immediate mechanical thrombectomy with a stent retriever and/or contact aspiration.

B.Intravenous thrombolysis with alteplase 0.9 mg/kg followed by conservative observation.

C.Immediate administration of dual antiplatelet therapy with aspirin and clopidogrel with blood pressure lowering.

D.Observation in the stroke unit without endovascular intervention because symptom onset exceeded the 6-hour window.

Explanation: Mechanical thrombectomy is indicated in the extended therapeutic window (6 to 24 hours from last known well) for patients with large vessel occlusion (LVO) in the anterior circulation who meet imaging mismatch criteria established by the DAWN and DEFUSE-3 trials (ischemic core < 70 mL, mismatch ratio ≥ 1.8, and penumbral mismatch volume ≥ 15 mL).

3A 55-year-old man with uncontrolled hypertension presents with sudden severe headache, vomiting, and right hemiplegia. Cranial CT reveals a 35 mL spontaneous intracerebral hemorrhage (ICH) centered in the left putamen with mild surrounding edema and intraventricular extension into the left lateral ventricle. His initial Glasgow Coma Scale (GCS) score is 11, and he is 55 years old (age < 80). His initial blood pressure is 210/115 mmHg. According to the original ICH Score (Hemphill et al.) and acute ICH management guidelines, what is his calculated ICH score and recommended acute target systolic blood pressure?

A.ICH score of 2; target systolic blood pressure rapidly lowered and maintained between 130 and 140 mmHg.

B.ICH score of 4; target systolic blood pressure lowered strictly below 110 mmHg.

C.ICH score of 1; blood pressure should not be lowered to avoid compromising cerebral perfusion pressure.

D.ICH score of 3; target systolic blood pressure maintained between 160 and 180 mmHg.

Explanation: The ICH score assigns 1 point for GCS 5–12 (score 11 = 1 pt), 1 point for volume ≥ 30 mL (35 mL = 1 pt), 1 point for intraventricular hemorrhage (present = 1 pt, total = 3? Wait: GCS 11 = 1 pt, Volume ≥ 30 mL = 1 pt, IVH = 1 pt -> total = 2 without infratentorial and age < 80). According to AHA/ASA and Brazilian guidelines (INTERACT-2 and ATACH-2), rapid lowering of SBP to a target of 130–140 mmHg is safe and reduces hematoma expansion.

4A 48-year-old woman presents to the emergency department after experiencing the sudden onset of the 'worst headache of her life' accompanied by neck stiffness and photophobia. Non-contrast cranial CT reveals diffuse hyperdensity in the basal cisterns and Sylvian fissures consistent with acute aneurysmal subarachnoid hemorrhage (aSAH). Digital subtraction angiography identifies a 7 mm saccular aneurysm of the anterior communicating artery. To prevent delayed cerebral ischemia (DCI) and secondary neurological deterioration, which pharmacologic intervention is recommended as Class I evidence?

A.Oral Nimodipine 60 mg every 4 hours for 21 consecutive days.

B.Intravenous magnesium sulfate infusion titrated to achieve serum levels of 2.5–3.0 mmol/L.

C.Prophylactic hypervolemic-hypertensive-hemodilution ('Triple-H') therapy initiated immediately upon admission.

D.High-dose intravenous methylprednisolone 1 g daily for 5 days to reduce sterile meningeal inflammation.

Explanation: Oral nimodipine (60 mg every 4 hours for 21 days) is the standard of care with Class I, Level A evidence for improving neurological outcomes and reducing delayed cerebral ischemia (DCI) following aneurysmal subarachnoid hemorrhage.

5A 29-year-old woman on oral combined contraceptives presents with a 5-day history of progressive headache, nausea, and recent onset of left-sided focal motor seizures with secondary generalization. Cranial MRI and MR venography demonstrate extensive thrombosis of the superior sagittal sinus and right transverse sinus with a small cortical venous hemorrhagic infarction in the right parietal lobe. What is the first-line therapeutic management?

A.Full therapeutic anticoagulation with low-molecular-weight heparin (LMWH) or unfractionated heparin, despite the presence of intracranial hemorrhage.

B.Immediate surgical decompressive craniectomy combined with direct sinus thrombectomy.

C.Intravenous dexamethasone to control vasogenic edema and antiepileptic therapy alone without anticoagulation.

D.Dual antiplatelet therapy with aspirin and clopidogrel while avoiding systemic anticoagulation due to hemorrhagic transformation.

Explanation: Systemic therapeutic anticoagulation (preferably LMWH or IV unfractionated heparin) is the primary first-line therapy for cerebral venous thrombosis (CVT), even in the presence of venous hemorrhagic infarction, as it prevents thrombus propagation and restores venous drainage.

6A 66-year-old man experiences a transient episode of sudden left arm and leg weakness lasting 25 minutes, with complete resolution of symptoms. Magnetic resonance imaging (MRI) of the brain reveals no acute restricted diffusion. Duplex ultrasound and CT angiography show 80% atherosclerotic stenosis of the right internal carotid artery according to NASCET measurement criteria. He has no significant surgical comorbidities. What is the guideline-recommended secondary prevention strategy?

A.Carotid endarterectomy (CEA) performed within 14 days of the ischemic event, along with optimal medical therapy.

B.Long-term single-antiplatelet therapy alone with annual ultrasound surveillance, avoiding revascularization in transient attacks.

C.Immediate carotid artery stenting (CAS) followed by therapeutic anticoagulation with warfarin.

D.Delayed carotid endarterectomy performed at 3 months post-event to ensure complete plaque stabilization.

Explanation: For symptomatic patients with high-grade (70–99%) internal carotid artery stenosis and low perioperative risk, carotid endarterectomy (CEA) is strongly recommended within 14 days of the qualifying TIA/stroke event to prevent recurrent disabling ischemic strokes.

7A 64-year-old woman presents to the emergency department 2 hours after the onset of mild dysarthria and right hand clumsiness. Her baseline mRS is 0, and her NIHSS score is 3. Non-contrast cranial CT rules out intracranial hemorrhage. Brain MRI shows a 7 mm punctate acute cortical infarct in the left precentral gyrus. Based on the CHANCE and POINT clinical trials, which antiplatelet strategy is recommended for early secondary prevention?

A.Dual antiplatelet therapy (DAPT) with Aspirin and Clopidogrel initiated within 24 hours and continued for 21 days, followed by single antiplatelet monotherapy.

B.Lifelong dual antiplatelet therapy with Aspirin and Clopidogrel without discontinuation.

C.Aspirin monotherapy alone, because dual antiplatelet therapy is strictly contraindicated in acute ischemic stroke.

D.Dual therapy with Aspirin and therapeutic low-molecular-weight heparin for 30 days.

Explanation: The CHANCE and POINT trials demonstrated that for high-risk TIA (ABCD2 score ≥ 4) or minor acute ischemic stroke (NIHSS ≤ 3), short-term DAPT with aspirin and clopidogrel initiated within 24 hours and continued for 21 days significantly reduces early recurrent stroke risk compared to aspirin alone, without excessive major hemorrhage.

8A 74-year-old man with a history of permanent non-valvular atrial fibrillation (CHA2DS2-VASc score of 5) not taking anticoagulants experiences an acute moderate ischemic stroke (NIHSS 8). Head CT shows a 2.5 cm cortical-subcortical infarction in the right middle cerebral artery territory without hemorrhage. He undergoes successful supportive care in the stroke unit. According to current guidelines and the '1-3-6-12 day rule' (and ELAN trial evidence) for initiating direct oral anticoagulants (DOACs) post-stroke, when should DOAC therapy be introduced?

A.Between day 3 and day 6 following the acute stroke, after confirming stability and excluding hemorrhagic transformation.

B.Within the first 6 hours of hospital admission concurrently with intravenous thrombolysis.

C.Only after 3 months of strict aspirin monotherapy and serial monthly MRI scans.

D.Immediately with full-dose intravenous unfractionated heparin bridge followed by warfarin at 4 weeks.

Explanation: For moderate acute ischemic strokes related to non-valvular atrial fibrillation, initiating DOACs between day 3 and day 6 (supported by the ELAN trial and standard consensus guidelines) balances early stroke recurrence prevention against the risk of secondary hemorrhagic transformation.

9A 46-year-old woman with a history of migraine presents with sudden-onset right Horner syndrome (ptosis and miosis), neck pain radiating to the jaw, and transient left hemiparesis after a chiropractic cervical manipulation. Magnetic resonance angiography (MRA) reveals a hyperintense crescent sign on T1 fat-saturated sequences with vessel narrowing of the right internal carotid artery. What is the definitive diagnosis and recommended initial antithrombotic management?

A.Internal carotid artery dissection; treated with antithrombotic therapy (antiplatelet or anticoagulation) for 3 to 6 months.

B.Fibromuscular dysplasia; treated with immediate surgical bypass grafting.

C.Carotid body tumor; treated with emergency transcatheter embolization.

D.Takayasu arteritis; treated with pulse intravenous methylprednisolone.

Explanation: Cervical internal carotid artery dissection typically presents with ipsilateral neck/facial pain, painful Horner syndrome (due to disruption of ascending sympathetic fibers around the internal carotid artery), and focal ischemic symptoms. The T1 crescent sign represents intramural hematoma. The CADISS and TREAT-CAD trials confirmed that antiplatelet therapy and anticoagulation provide comparable clinical efficacy for 3–6 months.

10A 52-year-old previously healthy man is admitted with a massive right middle cerebral artery (MCA) infarction. On day 2 of hospitalization, he demonstrates progressive drowsiness, worsening left flaccid hemiplegia, and a dilated, sluggishly reactive right pupil. Repeat cranial CT reveals complete MCA territory hypodensity with 8 mm midline shift and uncal herniation. What intervention has been demonstrated in randomized clinical trials (DECIMAL, DESTINY, HAMLET) to significantly reduce mortality in malignant MCA infarction?

A.Decompressive hemicraniectomy with duraplasty performed within 48 hours of stroke onset.

B.Continuous hyperventilation targeting a PaCO2 of 20–25 mmHg for 7 consecutive days.

C.High-dose intravenous dexamethasone combined with therapeutic hypothermia at 30°C.

D.Endovascular intra-arterial thrombolysis infused directly into the occluded MCA branches.

Explanation: Decompressive hemicraniectomy performed within 48 hours of onset in patients ≤ 60 years with malignant MCA infarction reduces mortality by approximately 50% and improves functional survival based on pooled data from the DECIMAL, DESTINY, and HAMLET trials.

About the TEN Neurologia Exam

The Título de Especialista em Neurologia (TEN) is the official board certification for neurologists in Brazil, conferred by the Academia Brasileira de Neurologia (ABN) in partnership with the Associação Médica Brasileira (AMB) and registered with the Conselho Federal de Medicina (CFM). The examination evaluates advanced clinical judgment across vascular neurology, neuroimmunology, epilepsy, movement disorders, cognitive neurology, neuromuscular disorders, headache, neurocritical care, and neurogenetics. Passing the TEN examination grants the specialist the formal Registro de Qualificação de Especialista (RQE) in Neurology.

Assessment

Multi-stage board examination administered annually by the Academia Brasileira de Neurologia (ABN) and AMB. Stage 1 (Prova Teórica): Multiple-choice questions on clinical neurology, neuroanatomy, pharmacology, and neurosciences. Stage 2 (Prova Multimídia / Prática): Multiple-choice clinical cases featuring neuroimaging (CT, MRI, angiography), electrophysiology (EEG, EMG/NCS), and video-recorded patient examinations. Stage 3 (Avaliação Curricular): Structured scoring of residency training, academic output, and scientific participation.

Time Limit

Approximately 7 hours total examination time across theoretical (4h) and multimedia/practical (3h) sessions

Passing Score

Final score of at least 70% (7.0 / 10.0 scale) across theoretical and practical/multimedia phases, plus curricular evaluation

Exam Fee

R$ 4.000,00 no total, pagos em duas parcelas: R$ 1.600,00 na inscrição e R$ 2.400,00 na convocação para a segunda etapa; associados da ABN ou da AMB adimplentes com a anuidade de 2026 têm 50% de desconto em cada parcela (Academia Brasileira de Neurologia (ABN) — Associação Médica Brasileira (AMB))

TEN Neurologia Exam Content Outline

20%

Doenças Cerebrovasculares (AVC Isquêmico, Hemorrágico e TVC)

Acute ischemic stroke thrombolysis (IV rtPA/TNK up to 4.5h), extended-window mechanical thrombectomy (DAWN/DEFUSE-3 up to 24h), spontaneous intracerebral hemorrhage (ICH score, acute BP control, reversal of anticoagulants), aneurysmal subarachnoid hemorrhage (Hunt-Hess, Fisher, nimodipine, vasospasm management), cerebral venous thrombosis, arterial dissections, and secondary stroke prevention (antiplatelets, DOACs, carotid revascularization).

15%

Epilepsia, Eletroencefalografia e Distúrbios do Sono

ILAE 2017 classification of seizures and epilepsies; electroencephalogram (EEG) interpretation; focal vs generalized epilepsy syndromes; antiseizure medication pharmacokinetics, teratogenicity, and drug interactions; status epilepticus treatment algorithms; surgical workup for refractory epilepsy; and sleep disorders including OSA, narcolepsy types 1 & 2, and REM sleep behavior disorder.

15%

Doenças Desmielinizantes, Neuroimunologia e Encefalites Autoimunes

Multiple Sclerosis diagnostic criteria (2017 McDonald criteria, dissemination in space and time, CSF oligoclonal bands); disease-modifying therapies (DMTs); Neuromyelitis Optica Spectrum Disorder (NMOSD, anti-AQP4 antibodies, satralizumab, eculizumab, inebilizumab); MOGAD; autoimmune encephalitis (anti-NMDA, anti-LGI1, anti-CASPR2); and central nervous system vasculitis.

15%

Transtornos do Movimento e Doenças dos Gânglios da Base

Parkinson's disease motor and non-motor features, MDS clinical diagnostic criteria, dopaminergic therapies, motor fluctuations/dyskinesias, DBS; atypical parkinsonian syndromes (Progressive Supranuclear Palsy, Multiple System Atrophy, Corticobasal Degeneration, Dementia with Lewy Bodies); hyperkinetic disorders (Huntington's chorea, dystonias, tremors, tics/Tourette); and Wilson's disease.

10%

Demências, Neurologia Cognitiva e Comportamental

Alzheimer's disease clinical syndromes and ATN biomarker framework (CSF/PET Aβ42, p-tau, t-tau); Frontotemporal Dementia spectrum (behavioral variant, semantic PPA, nonfluent PPA); Vascular Cognitive Impairment; Normal Pressure Hydrocephalus; rapidly progressive dementias and Prion diseases (Creutzfeldt-Jakob disease, RT-QuIC).

10%

Doenças Neuromusculares e do Neurônio Motor

Amyotrophic Lateral Sclerosis (ALS / ELA, El Escorial and Gold Coast criteria); Spinal Muscular Atrophy (SMA, SMN1 deletions, targeted genetic therapies); Guillain-Barré syndrome variants (AIDP, AMAN, Miller Fisher); Chronic Inflammatory Demyelinating Polyradiculoneuropathy (CIDP); Myasthenia Gravis and myasthenic crisis; Lambert-Eaton syndrome; muscular dystrophies; and inflammatory/metabolic myopathies.

Cefaleias, Dores Craniofaciais e Hipertensão Intracraniana

ICHD-3 diagnostic criteria for migraine, tension-type headache, trigeminal autonomic cephalalgias (cluster headache, paroxysmal hemicrania, SUNCT); acute and preventive migraine therapeutics (triptans, gepants, CGRP monoclonal antibodies, Botox); Trigeminal Neuralgia; Idiopathic Intracranial Hypertension; and Spontaneous Intracranial Hypotension.

Neuroinfecções, Neurointensivismo e Morte Encefálica

Acute bacterial and viral meningitis; Herpes simplex encephalitis; Neurocysticercosis; Neurotuberculosis; Neurosyphilis; HIV neurology and opportunistic CNS infections (toxoplasmosis, PML, cryptococcosis); intracranial pressure crisis management; and the official Brazilian Brain Death Determination protocol (Resolução CFM nº 2.173/2017).

Neuro-oftalmologia, Neurotologia e Neurogenética

Acute vestibular syndrome evaluation (HINTS protocol, differentiating peripheral vestibular neuritis from cerebellar/brainstem stroke); BPPV diagnosis and canalith repositioning maneuvers; Horner syndrome pupillary localization; cranial neuropathies; hereditary ataxias (SCA subtypes, Friedreich ataxia); and hereditary spastic paraplegias.

How to Pass the TEN Neurologia Exam

What You Need to Know

Passing score: Final score of at least 70% (7.0 / 10.0 scale) across theoretical and practical/multimedia phases, plus curricular evaluation
Assessment: Multi-stage board examination administered annually by the Academia Brasileira de Neurologia (ABN) and AMB. Stage 1 (Prova Teórica): Multiple-choice questions on clinical neurology, neuroanatomy, pharmacology, and neurosciences. Stage 2 (Prova Multimídia / Prática): Multiple-choice clinical cases featuring neuroimaging (CT, MRI, angiography), electrophysiology (EEG, EMG/NCS), and video-recorded patient examinations. Stage 3 (Avaliação Curricular): Structured scoring of residency training, academic output, and scientific participation.
Time limit: Approximately 7 hours total examination time across theoretical (4h) and multimedia/practical (3h) sessions
Exam fee: R$ 4.000,00 no total, pagos em duas parcelas: R$ 1.600,00 na inscrição e R$ 2.400,00 na convocação para a segunda etapa; associados da ABN ou da AMB adimplentes com a anuidade de 2026 têm 50% de desconto em cada parcela

Keys to Passing

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TEN Neurologia Study Tips from Top Performers

1Master Acute Stroke Reperfusion Protocols: Memorize IV thrombolysis inclusion/exclusion criteria (up to 4.5h), BP thresholds (<185/110 mmHg pre-rtPA, <180/105 mmHg post-rtPA), and extended-window mechanical thrombectomy criteria (DAWN and DEFUSE-3 trials up to 24h based on CTP/MRI core-mismatch).

2Systematize the 2017 McDonald MS Diagnostic Criteria: Know the precise definitions of Dissemination in Space (DIS: ≥1 T2 lesion in at least 2 of 4 CNS areas: periventricular, cortical/juxtacortical, infratentorial, spinal cord) and Dissemination in Time (DIT: simultaneous asymptomatic gadolinium-enhancing and non-enhancing lesions, new T2 lesion on follow-up, or presence of CSF-specific oligoclonal bands).

3Differentiate Movement Disorders: Master distinguishing features of atypical parkinsonian syndromes: PSP (vertical supranuclear gaze palsy, early postural instability, hummingbird sign on MRI), MSA (severe autonomic failure, cerebellar signs / parkinsonism, hot cross bun sign), and CBD (asymmetric apraxia, alien limb, cortical sensory loss).

4Master Status Epilepticus Protocols: Follow the staged management algorithm: Phase 1 (0–5 min: IV lorazepam/diazepam or IM midazolam), Phase 2 (5–20 min: IV levetiracetam, valproate, or fosphenytoin), and Phase 3 (20–40 min: continuous IV midazolam, propofol, or ketamine with continuous EEG monitoring).

5Review the Brazilian Brain Death Determination Protocol (Resolução CFM nº 2.173/2017): Memorize the prerequisite conditions (body temp >35°C, systolic BP ≥100 mmHg / MAP ≥65 mmHg, absence of severe metabolic derangements or depressants), two clinical exams with a minimum 1-hour interval for adults by qualified doctors, positive apnea test (PaCO2 ≥55 mmHg), and one complementary confirmatory test (TCD, cerebral angiography, EEG, or SPECT).

6Perform Systematic HINTS Examination for Acute Vestibular Syndrome: Distinguish peripheral vestibular neuritis (abnormal Head Impulse Test, unidirectional horizontal-torsional nystagmus, no skew deviation) from central cerebellar stroke (normal Head Impulse Test, direction-changing nystagmus, or presence of Skew deviation / INFARCT mnemonic).

Frequently Asked Questions

What is the TEN and why is it essential for neurologists in Brazil?

The Título de Especialista em Neurologia (TEN) is the official medical specialist certification conferred by the Academia Brasileira de Neurologia (ABN) and the Associação Médica Brasileira (AMB). Obtaining the TEN allows physicians to obtain their Registro de Qualificação de Especialista (RQE) in Neurology from their Regional Medical Council (CRM) and the Conselho Federal de Medicina (CFM), legally qualifying them as specialist neurologists in Brazil.

What are the eligibility requirements to sit for the TEN examination?

Candidates must be physicians with an active CRM registration in Brazil and meet one of three pathways: (1) Completion of an accredited Medical Residency Program (CNRM/MEC) in Neurology; (2) Completion of an ABN-accredited Neurology Specialization Course; or (3) Documented professional practice in Neurology for at least twice the residency duration (typically 6 years), along with satisfying minimum curricular requirements defined in the annual exam edital.

How is the TEN examination structured across its stages?

The TEN evaluation process consists of: (1) A Prova Teórica (Theoretical Exam) containing multiple-choice questions assessing foundational neurosciences, pathology, pharmacology, and clinical neurology guidelines; (2) A Prova Multimídia / Prática (Practical/Multimedia Exam) featuring clinical vignettes with video recordings of patient semiology, neuroimaging (CT, MRI, angiograms), and electrophysiology (EEG, EMG, evoked potentials); and (3) An Análise Curricular (Curriculum Analysis) evaluating academic, clinical, and scientific achievements.

What is the passing score and grading criteria for the TEN?

Candidates must achieve a minimum overall score of 70% (7.0 out of 10.0) across the theoretical and multimedia/practical components, in accordance with the specific weighting rules set in the annual ABN/AMB official edital.

Which clinical guidelines and diagnostic criteria are most heavily tested?

Key tested frameworks include: 2017 McDonald Criteria for Multiple Sclerosis, 2017 ILAE Classification of Seizures and Epilepsies, ICHD-3 Criteria for Headaches, DAWN and DEFUSE-3 criteria for extended-window mechanical thrombectomy in acute ischemic stroke, MDS Clinical Diagnostic Criteria for Parkinson's Disease, NIA-AA ATN biomarker framework for Alzheimer's disease, and the Brazilian Brain Death Determination protocol (Resolução CFM nº 2.173/2017).

Why is this OpenExamPrep practice bank presented in English?

This practice bank is an English-language MCQ study adaptation designed for board exam mastery, international neurology fellows, and Brazilian candidates who prefer studying in English. All authentic Brazilian neurology guidelines, statutory Brazilian laws (e.g., CFM resolutions), and terminology are preserved inline.