100+ Free TED — Título de Especialista em Dermatologia Practice Questions

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2026 Statistics

Key Facts: TED — Título de Especialista em Dermatologia Exam

120 Total Items

Official Exam Questions (80 MCQ + 40 Image Items)

Edital Oficial TED 2026 — SBD/AMB

60% in Each Phase

Independent Passing Cutoff

Regulamento SBD/AMB

4h + 3.5h

Exam Duration (Phase 1 & Phase 2)

Edital TED SBD

R$ 1.600 - R$ 3.200

Examination Registration Fee

Edital SBD/AMB 2026

RQE Dermatologia

CFM/CRM Specialist Qualification Granted

Conselho Federal de Medicina

60º Exame

Official 2026 Edition Number

Sociedade Brasileira de Dermatologia

The TED (SBD/AMB) 2026 board exam is a two-phase written specialty examination comprising an 80-question multiple-choice theoretical exam (Phase 1) and a 40-item image-projected written diagnostic exam (Phase 2). Passing requires achieving at least 60% on EACH written phase. Key subject pillars include clinical dermatology, cutaneous oncology, tropical infections/hanseníase, autoimmune bullous diseases, inflammatory dermatoses, dermatopathology, trichology, onychology, pediatric genodermatoses, and dermatologic surgery.

Sample TED — Título de Especialista em Dermatologia Practice Questions

Try these sample questions to test your TED — Título de Especialista em Dermatologia exam readiness. Each question includes a detailed explanation. Start the interactive quiz above for the full 100+ question experience with AI tutoring.

1During the dermatological physical examination of a patient with a generalized eruption, the examiner observes the formation of an epidermal detachment upon applying lateral tangential shearing pressure to normal-appearing perilesional skin. Which clinical sign and associated pathophysiological mechanism are demonstrated?

A.Nikolsky sign (sinal de Nikolsky), indicating loss of keratinocyte-keratinocyte cell adhesion (acantholysis) or complete basal-suprabasal junctional failure.

B.Asboe-Hansen sign, indicating lateral accumulation of edema within the subcorneal space under mechanical occlusion.

C.Darier sign, indicating sudden degranulation of dermal mastocytes with histamine release upon mechanical stroking.

D.Auspitz sign, indicating exposure of dilated and tortuous dermal papillary capillaries following mechanical removal of adherent parakeratotic scales.

Explanation: The direct Nikolsky sign (sinal de Nikolsky) is elicited by applying gentle tangential shearing friction with a finger on normal-appearing skin adjacent to lesions, producing epidermal detachment or cleavage. It reflects severe loss of intraepidermal keratinocyte cohesion (acantholysis, as seen in Pemphigus Vulgaris and Pemphigus Foliaceus) or extensive dermoepidermal necrolysis (as seen in Toxic Epidermal Necrolysis and Staphylococcal Scalded Skin Syndrome).

2A 32-year-old male presents with reddish-brown macules and papules on the trunk. Stroking one of the lesions with a blunt wooden spatula produces rapid local swelling, erythema, and an urticarial wheal confined to the stroked lesion within 5 minutes. What is this diagnostic phenomenon, and which condition does it confirm?

A.Auspitz sign, pathognomonic of guttate psoriasis.

B.Darier sign (sinal de Darier), pathognomonic of cutaneous mastocytosis (urticaria pigmentosa).

C.Koebner isomorphic phenomenon, indicative of lichen planus.

D.Hutchinson sign, indicative of subungual melanoma progression.

Explanation: The Darier sign (sinal de Darier) is the formation of a localized urticarial wheal and flare response following gentle mechanical friction or stroking of a pigmented macule or plaque. It is caused by the mechanical degranulation of clustered mast cells in the dermis with rapid release of histamine and vasoactive mediators, serving as a classic clinical hallmark of cutaneous mastocytosis (mastocitose cutânea / urticária pigmentosa).

3Which of the following describes the Koebner isomorphic phenomenon (fenômeno isomórfico de Koebner) and correctly lists dermatological diseases where it is classically observed?

A.Immediate wheal and erythema appearing within seconds of scratching normal skin, typical of symptomatic dermographism and acute cholinergic urticaria.

B.Spontaneous peripheral enlargement of flaccid bullae upon vertical digital pressure, seen in bullous pemphigoid and porphyria cutanea tarda.

C.Appearance of typical disease-specific isomorphic lesions at previously unaffected sites following non-specific mechanical, physical, or chemical trauma, characteristically seen in Psoriasis, Lichen Planus, and Vitiligo.

D.Development of pinpoint capillary hemorrhage beneath silver-white scales during curettage, typical of pityriasis rubra pilaris.

Explanation: The Koebner isomorphic phenomenon (fenômeno isomórfico de Koebner) is the occurrence of morphologically identical, active disease lesions in previously uninvolved skin following various forms of cutaneous trauma (e.g., scratching, surgical incisions, sunburn, or friction). It is classically documented in Psoriasis, Lichen Planus, and Vitiligo, and can also be observed in other conditions such as Kaposi sarcoma and verruca plana.

4A 45-year-old female presents with multiple yellowish-brown translucent papules and plaques on the midface. Diascopy (vitropressão) is performed by pressing a transparent glass slide firmly against the active infiltrated border of a lesion. The examiner notes the persistence of discrete 'apple-jelly' (geléia de maçã) yellowish-brown translucent nodules. This semiological sign strongly suggests which histopathological finding?

A.Accumulation of lipid-laden histiocytes in the upper reticular dermis (Touton giant cells).

B.Dense subepidermal band-like infiltrate of CD8+ T lymphocytes with colloid Civatte bodies.

C.Subcorneal collections of neutrophils with complete epidermal acantholysis.

D.Dermal epithelioid non-caseating or caseating granulomas with clearance of background erythema and telangiectasia.

Explanation: Diascopy (vitropressão) empties blood from dermal capillaries, eliminating transient erythema and unmasking underlying extravascular pigment or dermal cellular infiltration. The persistent 'apple-jelly' (aspecto em geléia de maçã) translucent yellowish-brown appearance is characteristic of epithelioid dermal granulomatous infiltrates, famously observed in cutaneous sarcoidosis, lupus vulgaris (cutaneous tuberculosis), and cutaneous leishmaniasis.

5In dermoscopy of non-melanocytic cutaneous lesions, which set of dermoscopic structures is most specific and characteristic for a pigmented Seborrheic Keratosis (ceratose seborreica)?

A.Comedo-like openings (crypts), milia-like cysts, hairpin vessels with white halos, and brain-like / gyri-and-sulci pattern.

B.Arborizing telangiectasias, blue-gray ovoid nests, and ulceration.

C.Atypical pigment network with thick lines, irregular dots/globules at the periphery, and blue-white veil.

D.Crown vessels surrounding a central yellowish-white umbilicated keratinaceous core.

Explanation: The dermoscopic hallmark of seborrheic keratosis (ceratose seborreica) comprises comedo-like openings (pseudocomedões or keratin-filled crypts), milia-like cysts (pseudocistos de mílio), hairpin vessels typically surrounded by a pale halo, moth-eaten borders, and a cerebriform or 'gyri and sulci' surface architecture (aspecto em cérebro / fissuras e cristas).

6A 68-year-old male presents with the sudden eruptive appearance of hundreds of pruritic seborrheic keratoses on his back over a 4-week period. He also reports an unintentional 8 kg weight loss. What paraneoplastic sign is present, and what is its most common underlying visceral malignancy?

A.Bazex syndrome, most commonly associated with squamous cell carcinoma of the upper aerodigestive tract.

B.Sign of Leser-Trélat, most commonly associated with adenocarcinoma of the gastrointestinal tract (especially gastric adenocarcinoma).

C.Sweet syndrome, most commonly associated with acute myeloid leukemia.

D.Necrolytic migratory erythema, most commonly associated with a glucagon-secreting pancreatic alpha-cell tumor.

Explanation: The sign of Leser-Trélat (sinal de Leser-Trélat) is characterized by the sudden eruption of multiple, rapidly growing, pruritic seborrheic keratoses, often on an inflammatory base. It represents an obligate/facultative cutaneous paraneoplastic syndrome driven by tumor-secreted growth factors (notably TGF-alpha and EGF), most frequently linked to gastrointestinal adenocarcinomas (particularly gastric adenocarcinoma), followed by colonic and hematologic malignancies.

7A 52-year-old man with chronic hepatitis C and heavy alcohol consumption presents with skin fragility, tense bullae, erosions, and milia on the dorsum of both hands, alongside facial hypertrichosis and brownish-purple urine. Wood's lamp examination of his urine reveals bright coral-pink fluorescence. Which enzyme deficiency causes this condition, and what is the definitive diagnosis?

A.Ferrochelatase deficiency; Erythropoietic Protoporphyria.

B.Porphobilinogen deaminase deficiency; Acute Intermittent Porphyria.

C.Uroporphyrinogen decarboxylase (UROD) deficiency; Porphyria Cutanea Tarda (PCT).

D.Uroporphyrinogen III synthase deficiency; Congenital Erythropoietic Porphyria (Günther disease).

Explanation: Porphyria Cutanea Tarda (PCT / Porfiria Cutânea Tardia) is caused by a deficiency in uroporphyrinogen decarboxylase (UROD), either acquired (sporadic type I, often triggered by hepatitis C, alcohol, iron overload, or estrogens) or inherited (familial type II). It presents with extreme mechanical skin fragility, blisters, milia, and hyperpigmentation on sun-exposed extremities, facial hypertrichosis, and pink-coral urinary fluorescence under Wood's lamp due to high uroporphyrin concentrations.

8In cutaneous semiology, which morphological definition precisely characterizes a 'lichenification' (liquenificação)?

A.Transient, circumscribed edematous elevation of the dermis with pale center and erythematous border lasting less than 24 hours.

B.Loss of substance involving both the epidermis and the full thickness of the dermis, healing with residual scar formation.

C.Thinning of the epidermis and dermis with loss of skin markings and a cigarette-paper-like wrinkling appearance.

D.Circumscribed thickening of the skin characterized by hyperkeratosis, acanthosis, and prominence of natural skin markings (quadrillage cutâneo), caused by chronic repeated rubbing or scratching.

Explanation: Lichenification (liquenificação) is a secondary elementary skin lesion characterized by cutaneous thickening, hyperpigmentation, and pronounced accentuation of normal skin surface markings (quadrillage cutâneo / acentuação dos sulcos normais). It is the classic anatomical result of chronic, repetitive scratching or mechanical rubbing in pruritic diseases such as atopic dermatitis and lichen simplex chronicus.

9A 48-year-old female presents with recurrent episodes of painful oral aphthae, genital ulcerations, erythema nodosum-like lesions on the lower legs, and sterile pustules arising at the site of venous puncture needle pricks within 24–48 hours. What diagnostic test is positive, and what is the underlying condition?

A.Pathergy test (teste de patergia); Behçet disease (Doença de Behçet).

B.Montenegro intradermal test; American tegumentary leishmaniasis.

C.Mitsuda test; Tuberculoid leprosy.

D.Tzanck smear; Recurrent Herpes Simplex Virus infection.

Explanation: The pathergy phenomenon (fenômeno de patergia) is a state of altered tissue reactivity where minor non-specific trauma (such as an oblique puncture with a sterile 20-gauge needle) induces an aseptic erythematous papule or pustule within 24 to 48 hours. It is an established criterion for Behçet disease (Doença de Behçet), which classically features recurrent bipolar oral/genital aphthae, uveitis, and cutaneous vasculitic manifestations.

10Which of the following primary cutaneous lesions is defined as a solid, elevated, palpable lesion greater than 1.0 cm in diameter that extends deeper into the dermis or subcutaneous tissue?

A.Macule (mácula).

B.Nodule (nódulo).

C.Vesicle (vesícula).

D.Scale (escama).

Explanation: In standard dermatological semiology, a nodule (nódulo) is a solid, palpable, circumscribed, elevated or deeply seated lesion greater than 1.0 cm in diameter that involves the dermis and/or subcutaneous tissue (hipoderme).

About the TED — Título de Especialista em Dermatologia Exam

The Exame de Título de Especialista em Dermatologia (TED) is the premier qualifying board certification examination in Brazilian dermatology, organized jointly by the Brazilian Society of Dermatology (Sociedade Brasileira de Dermatologia - SBD) and the Brazilian Medical Association (Associação Médica Brasileira - AMB). Attaining the TED title confers official board certification and eligibility for specialist registration (Registro de Qualificação de Especialista - RQE) with the Federal and Regional Medical Councils (CFM/CRM). The examination evaluates advanced clinical acumen across all aspects of skin, hair, and nail pathology, cutaneous oncology, infectious and tropical diseases (notably Hanseníase/leprosy, leishmaniasis, and endemic mycoses), auto-immune and bullous conditions, basic dermatopathology, pediatric genodermatoses, and procedural dermatology. This practice set provides an English-language study adaptation containing 100 rigorous multiple-choice questions preserving authentic Brazilian clinical terms and SBD consensus guidelines.

Assessment

Two-phase examination run by the Sociedade Brasileira de Dermatologia with the AMB. 1ª Fase — Prova Teórica: 80 multiple-choice questions set against the conteúdo programático published in the edital. 2ª Fase — Prova Teórico-Prática: 40 items sat in 3 hours 30 minutes, built largely on clinical and dermatoscopic images. Both phases are written; neither is a hands-on station examination.

Time Limit

2ª Fase Prova Teórico-Prática: 3 hours 30 minutes; the 1ª Fase duration is set by each edition's edital

Passing Score

At least 60% in EACH of the two phases

Exam Fee

Set annually by the SBD edital and varies with SBD/AMB membership status; consult the current edital before budgeting (Sociedade Brasileira de Dermatologia (SBD) — Associação Médica Brasileira (AMB))

TED — Título de Especialista em Dermatologia Exam Content Outline

20%

Dermatologia Clínica e Semiologia Cutânea

Morphological classification of elementary lesions (macules, papules, plaques, nodules, vesicles, bullae, wheals, scales, crusts, lichenification, atrophy), cutaneous semiology signs (Nikolsky, Asboe-Hansen, Darier, Auspitz, Koebner, dermographism), dermoscopy principles for pigmented and non-pigmented lesions, photobiology, and systemic manifestations of skin disorders.

18%

Oncologia Cutânea e Tumores Benignos

Basal cell carcinoma (nodular, superficial, morpheaform, infiltrative), squamous cell carcinoma and keratoacanthoma, cutaneous melanoma (superficial spreading, nodular, lentigo maligna, acral lentiginous; Breslow depth, ulceration, AJCC 8th edition staging, sentinel lymph node biopsy indications, targeted therapies BRAF/MEK inhibitors, immune checkpoint inhibitors), cutaneous lymphomas (Mycosis Fungoides, Sézary syndrome, CD30+ lymphoproliferative disorders), Kaposi sarcoma, and Merkel cell carcinoma.

18%

Infecciosas, Hanseníase e Dermatologia Tropical

Hanseníase / Hansen's disease (Ministry of Health / WHO operational classifications, Madrid and Ridley-Jopling spectrum, bacilloscopy index, histopathology, Mitsuda intradermal test, Type 1 reversal reactions and Type 2 Erythema Nodosum Leprosum reactions, PQT-U multidrug therapy regimen, and thalidomide/corticosteroid management), American tegumentary leishmaniasis, subcutaneous and deep mycoses (paracoccidioidomycosis, sporotrichosis, chromoblastomycosis, mycetoma, lobomycosis), superficial mycoses, bacterial infections, viral dermatoses, and sexually transmitted infections (syphilis, lymphogranuloma venereum, donovanosis, chancroid).

14%

Dermatoses Inflamatórias, Erupções Medicamentosas e Reativas

Psoriasis pathophysiology, clinical forms (vulgaris, guttate, inverse, pustular, erythrodermic), PASI evaluation, phototherapy, classic systemics (methotrexate, acitretin, cyclosporine), and targeted biologics (anti-TNF, anti-IL12/23, anti-IL17, anti-IL23), atopic dermatitis (Hanifin-Rajka criteria, dupilumab, JAK inhibitors), lichen planus, pityriasis rubra pilaris, acne vulgaris and rosacea, and severe adverse drug reactions (DRESS syndrome, SJS/TEN, AGEP, fixed pigmented erythema).

12%

Doenças Autoimunes do Tecido Conectivo e Dermatoses Bolhosas

Systemic and cutaneous lupus erythematosus (discoid, subacute, tumid, acute; autoantibody profiles), dermatomyositis (heliotrope rash, Gottron papules, malignancy screening), systemic sclerosis (diffuse vs. limited/CREST), morphea, pemphigus vulgaris (anti-desmoglein 3 and 1, suprabasal acantholysis, tombstone basal layer), endemic and non-endemic pemphigus foliaceus / fogo selvagem (anti-desmoglein 1, subcorneal cleavage), bullous pemphigoid (anti-BP180 NC16A and anti-BP230, subepidermal blister, eosinophil infiltrate), dermatitis herpetiformis (anti-tissue transglutaminase, granular IgA in dermal papillae), and direct/indirect immunofluorescence patterns.

Dermatopatologia Básica e Padrões Histopatológicos

Fundamental tissue reaction patterns: spongiotic, psoriasiform, interface (vacuolar and lichenoid), vesiculobullous (intraepidermal vs. subepidermal), granulomatous (sarcoidal, palisading/necrobiotic, tuberculoid, suppurative), and panniculitis (septal without vasculitis, lobular with vasculitis). Mastery of special histochemical stains including PAS, Grocott-GMS, Ziehl-Neelsen, Fite-Faraco, Masson-Fontana, Perls, and Congo Red.

Tricologia, Onicologia, Genodermatoses e Dermatologia Pediátrica

Hair follicle biology, non-scarring alopecias (androgenetic alopecia, alopecia areata, telogen effluvium), scarring alopecias (lichen planopilaris, frontal fibrosing alopecia, central centrifugal cicatricial alopecia, discoid lupus), nail anatomy and pathology (trachyonychia, onychomycosis, nail psoriasis, longitudinal melanonychia, subungual melanoma), genodermatoses (neurofibromatosis type 1, tuberous sclerosis, xeroderma pigmentosum, hereditary epidermolysis bullosa subtypes, ichthyoses), and pediatric vascular anomalies (infantile hemangioma vs. vascular malformations).

Cirurgia Dermatológica, Cosmiatria e Laser

Surgical planning, lines of minimal tension (Langer lines), elliptical excisions, reconstructive flaps (advancement, rotation, transposition, interpolation) and skin grafts, Mohs micrographic surgery principles, local anesthetics pharmacology (lidocaine maximum doses with/without vasoconstrictor, signs of systemic toxicity, tumescent anesthesia), cryosurgery with liquid nitrogen, chemical peels, botulinum toxin application anatomy and adverse events, hyaluronic acid injection depth, rheology, vascular occlusion warning signs and hyaluronidase rescue protocols, and laser tissue interaction principles (selective photothermolysis, fractional lasers, Q-switched lasers).

How to Pass the TED — Título de Especialista em Dermatologia Exam

What You Need to Know

Passing score: At least 60% in EACH of the two phases
Assessment: Two-phase examination run by the Sociedade Brasileira de Dermatologia with the AMB. 1ª Fase — Prova Teórica: 80 multiple-choice questions set against the conteúdo programático published in the edital. 2ª Fase — Prova Teórico-Prática: 40 items sat in 3 hours 30 minutes, built largely on clinical and dermatoscopic images. Both phases are written; neither is a hands-on station examination.
Time limit: 2ª Fase Prova Teórico-Prática: 3 hours 30 minutes; the 1ª Fase duration is set by each edition's edital
Exam fee: Set annually by the SBD edital and varies with SBD/AMB membership status; consult the current edital before budgeting

Keys to Passing

Work through all 100 available questions
Review every answer and explanation
Track weak areas and revisit them
Use our AI tutor for tough concepts

TED — Título de Especialista em Dermatologia Study Tips from Top Performers

1Master the Diagnostic Algorithms for Hanseníase: Memorize the Madrid and Ridley-Jopling classifications, bacilloscopic index interpretations, distinguishing Type 1 (reverse) from Type 2 (erythema nodosum leprosum) reactional states, and the 2021+ Ministry of Health unified multidrug therapy (PQT-U).

2Integrate Clinical, Dermoscopic, and Histopathologic Clues: Practice correlating clinical presentations with key histopathological signs (e.g., Wickham striae and Civatte bodies in lichen planus, tombstoning in pemphigus vulgaris, Munro microabscesses in psoriasis, and asteriod bodies in sporotrichosis).

3Memorize Cutaneous Oncology Margins and Staging: Know the exact surgical margins for melanoma based on Breslow thickness (in situ: 0.5-1.0 cm; <=1.0 mm: 1 cm; 1.01-2.0 mm: 1-2 cm; >2.0 mm: 2 cm) and indications for sentinel lymph node biopsy (pT1b, Breslow >=0.8 mm or <0.8 mm with ulceration).

4Understand Biologics and Targeted Therapies: Review mechanism of action, screening requirements (QuantiFERON/PPD, hepatitis serologies), and adverse effect profiles for anti-TNF, anti-IL17 (secukinumab, ixekizumab), anti-IL23 (guselkumab, risankizumab), anti-IL4R (dupilumab), and JAK inhibitors.

5Review Aesthetic Complication Protocols: Thoroughly understand the early recognition of hyaluronic acid vascular occlusion (livedoid pattern, blanching, severe pain) and the emergency high-dose pulsed hyaluronidase protocol.

Frequently Asked Questions

What is the Título de Especialista em Dermatologia (TED)?

The Título de Especialista em Dermatologia (TED) is the official medical board certification in Dermatology in Brazil, conferred by the Brazilian Society of Dermatology (SBD) in partnership with the Brazilian Medical Association (AMB) and registered with the Federal Medical Council (CFM).

How is the 60º Exame TED (2026) structured?

The 2026 examination consists of two written eliminatory phases: Phase 1 (1ª Fase - Prova Teórica) containing 80 multiple-choice questions over 4 hours, and Phase 2 (2ª Fase - Prova Teórico-Prática) containing 40 projected image-based written diagnosis questions over 3.5 hours. Both phases are written tests and require at least a 60% minimum passing score in each phase independently.

What are the eligibility prerequisites to sit for the TED exam?

Candidates must hold an active medical license (CRM) in Brazil and have completed either: (1) a full 3-year Medical Residency Program in Dermatology accredited by CNRM/MEC or SBD-accredited training service, or (2) proven equivalent dermatological clinical practice and continuous training according to the strict criteria defined in the current official SBD/AMB Edital.

What is the passing score required for the TED examination?

Candidates must score at least 60% in Phase 1 (Prova Teórica) AND at least 60% in Phase 2 (Prova Teórico-Prática). Scoring below 60% in either phase results in elimination.

Why does the TED exam heavily test Hanseníase (Hansen's disease) and tropical mycoses?

Brazil has one of the highest leprosy burdens globally, alongside endemic pockets of American tegumentary leishmaniasis, sporotrichosis, and paracoccidioidomycosis. Specialist dermatologists in Brazil are expected to possess complete expertise in clinical diagnosis, slit-skin smear bacilloscopy, histopathology, reactional state management, and Ministry of Health multidrug protocols (PQT-U).

Is this practice question bank in English or Portuguese?

This practice bank is an English-language study adaptation designed for comprehensive specialty preparation, preserving exact official Brazilian medical terminology inline (e.g., SBD, AMB, TED, Hanseníase, Fogo Selvagem, PQT-U, Glucantime) to ensure complete alignment with the official Brazilian syllabus.