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Free Practice Questions for Cambodia Medicine Exit Exam

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Exam Review

Key Facts: Cambodia Medicine Exit Exam Exam

Sub-Decree 21

ANKr.BK of 13 March 2007 requiring national entrance and graduation examinations in health education

http://cambodiancouncilofnurse.com/wp-content/uploads/2017/05/Joint-Prakas-on-National-Training-Curriculum-for-ADN.-Eng.pdf

MCQ + OSCE

Assessment methods named for health-profession national exit examinations in the World Bank SPESHP project paper

https://documents1.worldbank.org/curated/en/668861591063363140/pdf/Cambodia-Strengthening-Pre-Service-Education-System-for-Health-Professionals-Project.pdf

Free 100-question English MCQ practice for Cambodia's national medicine exit exam. Scope follows published Cambodia MD core competencies. Independent study aid, not an official NECHS paper.

Sample Cambodia Medicine Exit Exam Practice Questions

Try these sample questions to review concepts for the Cambodia Medicine Exit Exam exam. Each question includes a detailed explanation. Start the interactive quiz above for the full 100+ question experience with AI tutoring.

1A febrile forest worker in Mondulkiri has intraerythrocytic parasites on a thin blood film. Which Plasmodium species is most associated with severe malaria and typically shows banana-shaped gametocytes?
A.Plasmodium malariae
B.Plasmodium falciparum
C.Plasmodium ovale
D.Plasmodium vivax
Explanation: Plasmodium falciparum causes almost all severe malaria deaths and is identified on thin films by multiply infected red cells, appliqué forms, and crescent (banana-shaped) gametocytes. P. vivax and P. ovale produce round gametocytes and large, Schüffner-dotted red cells, while P. malariae produces band forms in normal-sized cells. Species identification still matters in Cambodia because falciparum disease can deteriorate within hours and because vivax additionally requires hypnozoite treatment.
2During Cambodia’s rainy season, a Phnom Penh household has larvae in uncovered ceramic water jars. Which mosquito genus transmits dengue virus?
A.Aedes
B.Anopheles
C.Culex only as the sole dengue vector
D.Phlebotomus
Explanation: Dengue viruses are transmitted by Aedes mosquitoes, principally Aedes aegypti, which breed in artificial containers of clean water and bite mainly during the day. Anopheles mosquitoes transmit malaria, not dengue. Distinguishing these vectors matters in Cambodia because dengue control targets household water storage, whereas malaria control focuses on forest exposure and insecticide-treated nets.
3A 32-year-old man with diabetic ketoacidosis has Na+ 136 mmol/L, Cl− 100 mmol/L, and HCO3− 8 mmol/L. What is the serum anion gap using AG = Na+ − (Cl− + HCO3−)?
A.12 mmol/L
B.20 mmol/L
C.28 mmol/L
D.36 mmol/L
Explanation: The serum anion gap equals sodium minus the sum of chloride and bicarbonate: 136 − (100 + 8) = 28 mmol/L. A gap this high indicates accumulation of unmeasured anions, which in this patient are ketoacids. Calculating the gap is the first step in separating high-gap metabolic acidosis (ketoacidosis, lactate, toxins) from hyperchloremic normal-gap acidosis.
4In the renin–angiotensin–aldosterone axis, which action of angiotensin II most directly raises blood pressure within minutes?
A.Blocking ADH release from the posterior pituitary
B.Dilating afferent arterioles exclusively
C.Inhibiting aldosterone synthase in the adrenal cortex
D.Arteriolar vasoconstriction via AT1 receptors
Explanation: Angiotensin II binds vascular AT1 receptors and causes arteriolar vasoconstriction, raising systemic vascular resistance within minutes. It also stimulates aldosterone secretion and ADH release, which expand volume over hours to days, but the immediate pressor effect is vasoconstriction. This physiology underpins ACE-inhibitor and ARB therapy for hypertension and heart failure.
5A 14-year-old with dengue becomes afebrile on day 5, then develops abdominal pain, hemoconcentration, and a rapidly falling platelet count. Which pathophysiologic event best explains this critical-phase picture?
A.Primary bacterial invasion of the pulmonary alveoli
B.Transient increase in vascular permeability with plasma leakage
C.Complete bone-marrow aplasia lasting several weeks
D.Acute tubular necrosis as the first and only dengue mechanism
Explanation: In dengue, the critical phase typically begins around defervescence, when a transient increase in capillary permeability allows plasma to leak into serosal cavities. Hemoconcentration with a concurrent platelet drop is the laboratory counterpart of that leak and is a WHO 2009 warning pattern. Supportive isotonic-fluid therapy, not antivirals, determines survival during this window.
6Why does untreated advanced HIV infection markedly increase the risk of progression from latent Mycobacterium tuberculosis infection to active tuberculosis?
A.Loss of CD4+ T-cell help impairs macrophage containment of bacilli within granulomas
B.HIV immediately sterilizes all granulomas so reactivation becomes impossible
C.HIV converts M. tuberculosis into a virus that no longer needs cell-mediated immunity
D.HIV raises neutrophil counts so high that granulomas cannot form
Explanation: Containment of M. tuberculosis depends on CD4+ T-cell–macrophage cooperation and TNF-supported granulomas. Progressive CD4 depletion in advanced HIV collapses that containment and allows bacilli to multiply, which is why TB remains a leading opportunistic disease in Cambodia. This immunology is also why people with HIV need both prompt TB evaluation and antiretroviral therapy rather than delayed ‘wait-and-see’ care.
7A woman with severe dengue develops oozing from venipuncture sites, a prolonged PT and aPTT, low fibrinogen, and elevated D-dimer. Which process best explains these findings?
A.Isolated vitamin K deficiency without clotting-factor consumption
B.Factor VIII deficiency inherited as haemophilia A presenting in adulthood
C.Disseminated intravascular coagulation with consumption of platelets and clotting factors
D.Spurious laboratory error because dengue never affects coagulation
Explanation: Simultaneous prolongation of PT and aPTT, hypofibrinogenemia, high D-dimer, and bleeding from puncture sites indicate disseminated intravascular coagulation, in which thrombin generation consumes platelets and factors while fibrin is degraded. Severe dengue can trigger this consumptive coagulopathy on top of plasma leakage. Recognition matters because treatment is supportive resuscitation and blood-product replacement, not intramuscular injections into a coagulopathic limb.
8After a carbohydrate meal, which hormone most directly promotes GLUT4-mediated glucose uptake into skeletal muscle and adipose tissue?
A.Glucagon
B.Insulin
C.Cortisol
D.Adrenaline
Explanation: Insulin is released from pancreatic beta cells in response to a glucose rise and translocates GLUT4 to the muscle and adipocyte membrane, lowering plasma glucose. Glucagon, cortisol, and adrenaline are counter-regulatory hormones that raise glucose. This is the physiologic basis for using insulin in type 1 diabetes and for the hyperglycemia of stress and infection.
9A 55-year-old man with painless jaundice has a markedly raised conjugated (direct) bilirubin, dark urine, and pale stools. Which mechanism best explains this pattern?
A.Isolated unconjugated hyperbilirubinemia from Gilbert syndrome
B.Intravascular haemolysis flooding the liver with unconjugated bilirubin only
C.Failure of hepatic bilirubin conjugation in a neonate with immature glucuronyl transferase
D.Extrahepatic biliary obstruction preventing conjugated bilirubin from reaching the intestine
Explanation: Conjugated bilirubin that cannot enter the gut refluxes into plasma and is excreted in urine (dark urine), while stools become pale because stercobilin is not formed. Painless jaundice with this pattern raises concern for malignant biliary obstruction, such as pancreatic head carcinoma. Unconjugated hyperbilirubinemia does not darken the urine because unconjugated bilirubin is albumin-bound and not filtered.
10A patient with cholera-like watery diarrhoea has pH 7.28, PaCO2 28 mmHg, Na+ 138 mmol/L, Cl− 116 mmol/L, and HCO3− 14 mmol/L. Which acid–base diagnosis is most accurate?
A.High anion-gap metabolic acidosis from ketoacid accumulation
B.Hyperchloremic normal anion-gap metabolic acidosis from gastrointestinal bicarbonate loss
C.Uncompensated respiratory acidosis from alveolar hypoventilation
D.Metabolic alkalosis from gastric acid loss
Explanation: The anion gap is 138 − (116 + 14) = 8 mmol/L, a normal gap, while bicarbonate is low and chloride is high. Profuse small-bowel diarrhoea loses bicarbonate-rich fluid, producing hyperchloremic metabolic acidosis; the low PaCO2 is respiratory compensation. Distinguishing this from high-gap acidosis matters because the treatment priority is volume and bicarbonate-containing rehydration, not a search for ketoacids or lactate as the primary process.

About the Cambodia Medicine Exit Exam Exam

Cambodia requires medical graduates of public and private programmes to sit a national exit examination under Sub-Decree 21 ANKr.BK. NECHS administers the sitting. ASEAN AJCCM materials list successful completion of the national exit exam among physician licensing requirements, together with Medical Council of Cambodia registration. The UHS Faculty of Medicine degree profile published through AUN TASEMed lists Cambodia MD core competencies (biomedical, behavioural, public health, communication, organisation/research, diagnosis, and patient care). Official item count, duration, fee, and pass mark are unpublished. This free bank is independent OpenExamPrep English MCQ study for those domains, not an official NECHS paper and not MCC registration.

Exam sponsor: National Examination Committee for Health Sector Training (NECHS). The requirements and fees below concern the certification or admission exam, separate from our free practice resources.

Assessment

National competency exit sitting in Khmer for medical graduates. Content follows Cambodia MD core competencies documented in the UHS/AUN TASEMed degree profile (scientific foundations and patient care). Official format includes written MCQ and OSCE components per SPESHP. This 100-question bank is an independent English MCQ study adaptation, not an OSCE simulation.

Time Limit

Not published as a single sitting duration

Passing Score

Not published as a numeric cut score for the medicine sitting

Exam / Certification Fees

Not published in Sub-Decree 21 or SPESHP documents

Exam sponsor website

Fees, eligibility, and exam policies can change. Confirm them with the exam sponsor before applying or paying.

Our practice resources: topics covered

We aim to reflect publicly available exam outlines and topic information in our study resources. Coverage, format, and difficulty may differ from the actual exam, and we cannot guarantee that every detail is accurate or current. Confirm exam requirements, fees, and policies with the official exam sponsor.

Official weight unpublished (15 practice questions)

Biomedical sciences

Scientific foundations for medical practice.

Official weight unpublished (15 practice questions)

Behavioural science, public health, and ethics

Population health, communication, and professionalism.

Official weight unpublished (20 practice questions)

Diagnosis and investigations

History, examination, and investigation selection.

Official weight unpublished (25 practice questions)

Internal medicine

Adult medical conditions including infections of public-health importance in Cambodia.

Official weight unpublished (15 practice questions)

Surgery, emergency, and critical care

Acute surgical and emergency recognition.

Official weight unpublished (10 practice questions)

Obstetrics, paediatrics, and community care

Maternal, child, and community presentations.

Preparing for the Cambodia Medicine Exit Exam Exam

What You Need to Know

  • Passing score: Not published as a numeric cut score for the medicine sitting
  • Assessment: National competency exit sitting in Khmer for medical graduates. Content follows Cambodia MD core competencies documented in the UHS/AUN TASEMed degree profile (scientific foundations and patient care). Official format includes written MCQ and OSCE components per SPESHP. This 100-question bank is an independent English MCQ study adaptation, not an OSCE simulation.
  • Time limit: Not published as a single sitting duration
  • Exam / certification fees: Not published in Sub-Decree 21 or SPESHP documents Official sources

Using Our Practice Resources

  • Work through all 100 available questions
  • Review every answer and explanation
  • Track weak areas and revisit them
  • Use our AI tutor for tough concepts

Cambodia Medicine Exit Exam: Suggested Study Strategy

1Prioritise diagnosis and internal medicine, including dengue, malaria, TB, and HIV knowledge used in Cambodian practice.
2MCQs can teach emergency recognition; they cannot replace OSCE or clinical-skills practice.
3Review Khmer clinical terms because the official sitting is in Khmer.

Frequently Asked Questions

Is Medical Council of Cambodia registration the same as this exam?

No. MCC registration and licensing are dossier and CPD processes under the 2016 Law on Regulation of Health Practitioners. ASEAN AJCCM materials list the national exit exam as a separate completed requirement for local physicians.

Is this a University of Health Sciences exam only?

No. Sub-Decree 21 requires national graduation examinations for public and private health educational institutions. UHS is one training institution, not the sole sitting identity.