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100+ Free Czech Dermatovenerology Board Examination Practice Questions

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Key Facts: Czech Dermatovenerology Board Examination Exam

5 years

Specialty residency training duration

Věstník MZ ČR educational program

500 CZK

Standard statutory examination fee

Nařízení vlády č. 324/2018 Sb.

>= 3

Board examiners on committee

Vyhláška č. 282/2019 Sb.

3 topics

Drawn theoretical oral questions

MZ ČR / IPVZ examination rules

2 retakes

Maximum allowable attempts

Zákon č. 95/2004 Sb.

The Czech specialty examination in Dermatovenerology (Atestační zkouška) is the statutory exit board governed by Zákon č. 95/2004 Sb. and Vyhláška č. 282/2019 Sb. The official assessment is an oral and practical board exam before an at least 3-member committee, with statutory fees under Nařízení vlády č. 324/2018 Sb. of 500 CZK (3,500 CZK for first retake, 5,000 CZK for second retake). This 100-question English practice bank adapts core curriculum topics into clinical vignettes for high-yield exam preparation.

Sample Czech Dermatovenerology Board Examination Practice Questions

Try these sample questions to test your Czech Dermatovenerology Board Examination exam readiness. Each question includes a detailed explanation. Start the interactive quiz above for the full 100+ question experience with AI tutoring.

1A 42-year-old man presents with well-demarcated erythematous plaques covered by silvery-white scales on the extensor surfaces of his elbows and knees. Auspitz sign is positive. Which histological feature is characteristic of this condition in the active stratum spinosum?
A.Munro microabscesses within the parakeratotic stratum corneum
B.Spongiform pustules of Kogoj in the upper stratum spinosum
C.Civatte bodies at the dermoepidermal junction
D.Suprabasal acantholysis with a row-of-tombstones pattern
Explanation: Psoriasis vulgaris demonstrates spongiform pustules of Kogoj, which are collections of neutrophils within the upper stratum spinosum and stratum malpighii. Confluent parakeratosis, regular epidermal hyperplasia (acanthosis) with elongation of rete ridges, and thinning of suprapapillary plates with dilated tortuous capillaries in dermal papillae are also typical. Management of moderate-to-severe disease utilizes targeted biologics (anti-IL-17, anti-IL-23, or anti-TNF).
2A 35-year-old woman presents with intensely pruritic, polygonal, flat-topped, violaceous papules on the flexor aspects of her wrists. Close inspection reveals fine reticulated whitish lines on the surface (Wickham striae). What is the primary histopathological change responsible for this appearance?
A.Subcorneal blister formation with acantholytic cells
B.Band-like dense lymphocytic infiltrate at the dermoepidermal junction with saw-toothed rete ridges
C.Marked intercellular edema (spongiosis) throughout the epidermis with eosinophils
D.Leukocytoclasia with fibrinoid necrosis of postcapillary venules
Explanation: Lichen planus is characterized histologically by a dense, band-like (lichenoid) lymphocytic infiltrate hugging the dermoepidermal junction, basal cell vacuolization, wedge-shaped hypergranulosis, and saw-toothed irregular acanthosis. Apoptotic keratinocytes (Civatte or colloid bodies) are abundant at the junction. First-line management for localized cuticular lichen planus consists of superpotent topical corticosteroids (e.g., clobetasol propionate).
3A 48-year-old woman develops painful, flaccid bullae on normal-appearing skin and extensive oral erosions that bleed easily. Nikolsky sign is positive. Direct immunofluorescence (DIF) of perilesional skin demonstrates:
A.Linear IgG and C3 deposition along the epidermal basement membrane zone
B.Intercellular epidermal IgG and C3 deposition in a net-like or chicken-wire pattern
C.Granular IgA deposits clustered within the dermal papillae tips
D.Vascular wall deposition of IgM, IgG, and C3 in upper dermal vessels
Explanation: Pemphigus vulgaris features pathogenic autoantibodies targeting desmoglein 3 (and desmoglein 1 in mucocutaneous disease), producing intraepidermal suprabasal acantholysis. Direct immunofluorescence reveals intercellular IgG (predominantly IgG4) and C3 deposition between keratinocytes throughout the epidermis in a classic fishnet or chicken-wire pattern. First-line systemic treatment is rituximab combined with systemic corticosteroids.
4A 76-year-old man presents with widespread tense bullae on urticarial erythematous plaques involving the trunk and thighs. Nikolsky sign is negative, and the oral mucosa is spared. Serum ELISA detects autoantibodies against:
A.Desmoglein 1 and desmoglein 3
B.BP180 (NC16A domain) and BP230
C.Epidermal transglutaminase (eTG)
D.Type VII collagen within anchoring fibrils
Explanation: Bullous pemphigoid is an autoimmune subepidermal blistering disease driven by autoantibodies against hemidesmosomal antigens BP180 (specifically the NC16A domain) and BP230. Histopathology demonstrates a subepidermal blister containing prominent eosinophils, and direct immunofluorescence reveals continuous linear IgG and C3 along the basement membrane zone. First-line therapy includes superpotent topical corticosteroids (e.g., clobetasol cream applied to the whole body) or oral prednisone.
5A 32-year-old man presents with intensely burning and pruritic grouped vesicles, papules, and excoriations symmetrically distributed over his elbows, knees, and buttocks. Direct immunofluorescence of normal-appearing perilesional skin reveals granular IgA in the dermal papillae. Which intervention represents the definitive causal long-term therapy?
A.Oral dapsone without dietary changes
B.Strict lifelong gluten-free diet
C.Intravenous immunoglobulin (IVIG)
D.High-dose systemic oral methotrexate
Explanation: Dermatitis herpetiformis (Duhring disease) is the cutaneous manifestation of gluten-sensitive enteropathy (celiac disease), mediated by anti-epidermal transglutaminase (eTG) IgA autoantibodies that deposit in dermal papillae. While dapsone rapidly relieves pruritus and blistering within 24 to 48 hours, a strict lifelong gluten-free diet is the only definitive causal therapy that resolves cutaneous lesions, clears IgA deposits, and reverses gut enteropathy. Long-term dietary compliance eliminates the need for pharmacological therapy and reduces lymphoma risk.
6A 28-year-old woman develops well-demarcated annular polycyclic erythematous plaques with central clearing on sun-exposed areas of her neck and upper back. She has no scarring or follicular plugging. Serological testing is strongly positive for anti-Ro/SSA antibodies. What is the most likely diagnosis?
A.Chronic discoid lupus erythematosus (CDLE)
B.Subacute cutaneous lupus erythematosus (SCLE)
C.Systemic scleroderma (diffuse cutaneous SSc)
D.Dermatomyositis
Explanation: Subacute cutaneous lupus erythematosus (SCLE) presents as non-scarring, non-indurated papulosquamous or annular polycyclic lesions on sun-exposed skin (upper chest, back, shoulders, extensor arms). More than 80% of patients carry anti-Ro/SSA antibodies, and drug-induced etiologies (e.g., terbinafine, calcium channel blockers, PPIs, thiazides) must be ruled out. Photoprotection, topical corticosteroids/calcineurin inhibitors, and hydroxychloroquine constitute the standard therapeutic approach.
7A 52-year-old woman presents with violaceous periorbital edema, erythematous flat-topped papules overlying the dorsal metacarpophalangeal and interphalangeal joints, and progressive proximal muscle weakness. Which myositis-specific antibody is most strongly associated with rapidly progressive interstitial lung disease (RPILD) and amyopathic presentation?
A.Anti-Mi-2
B.Anti-MDA5 (melanoma differentiation-associated protein 5)
C.Anti-Jo-1 (histidyl-tRNA synthetase)
D.Anti-TIF1-gamma
Explanation: Anti-MDA5 autoantibodies in dermatomyositis are characteristically associated with clinically amyopathic or hypomyopathic dermatomyositis, cutaneous ulcerations, palmar violaceous papules, and a high risk of life-threatening, rapidly progressive interstitial lung disease (RPILD). Prompt initiation of combined immunosuppressive therapy (corticosteroids, calcineurin inhibitors, and cyclophosphamide) is crucial. Screening for internal malignancies and lung high-resolution CT are vital components of the diagnostic workup.
8A 45-year-old woman presents with Raynaud phenomenon, sclerodactyly, telangiectasias on her face and hands, calcinosis cutis, and esophageal dysphagia. Nailfold videocapillaroscopy reveals dilated giant capillaries with minimal capillary dropout. Which autoantibody profile is most characteristic of this limited cutaneous systemic sclerosis (CREST syndrome)?
A.Anti-topoisomerase I (anti-Scl-70)
B.Anticentromere antibodies (ACA / CENP-B)
C.Anti-RNA polymerase III
D.Anti-U1 RNP
Explanation: Limited cutaneous systemic sclerosis (lcSSc, formerly CREST syndrome) is characterized by skin sclerosis restricted to distal extremities (distal to elbows and knees) and face, prominent calcinosis, Raynaud, esophageal dysmotility, sclerodactyly, and telangiectasia. More than 70-80% of patients have anticentromere antibodies (ACA), which carry a lower risk of renal crisis and pulmonary fibrosis but higher risk of late-onset pulmonary arterial hypertension (PAH). Management focuses on organ-specific manifestations, vasodilators (calcium channel blockers, PDE-5 inhibitors), and regular echocardiographic screening for PAH.
9A 24-year-old man with severe, recalcitrant atopic dermatitis involving 60% of his body surface area (EASI score 32) fails to achieve disease control despite optimized topical corticosteroids and phototherapy. Which biologic agent targeting the interleukin-4 receptor alpha subunit (IL-4Rα) is indicated as first-line targeted systemic therapy?
A.Omalizumab
B.Dupilumab
C.Tralokinumab
D.Mepolizumab
Explanation: Dupilumab is a fully human monoclonal antibody directed against the shared alpha subunit of the interleukin-4 receptor (IL-4Rα), thereby inhibiting dual signaling of IL-4 and IL-13, the principal Type 2 cytokines driving atopic dermatitis. In clinical trials and international/Czech guidelines (ČDS ČLS JEP), dupilumab demonstrates robust efficacy in reducing pruritus, EASI scores, and restoring the skin barrier in moderate-to-severe AD. A well-known potential side effect requiring ophthalmologic vigilance is allergic/atopic conjunctivitis.
10A 30-year-old nurse develops itchy, erythematous, vesicular eczema on the dorsum of both hands. Allergic contact dermatitis to rubber accelerators in examination gloves is suspected. In standard European Baseline Series patch testing, at what intervals must readings be performed according to ESCD/Czech guidelines?
A.20 minutes and 2 hours after application
B.48 hours (after removal) and 72 to 96 hours
C.6 hours and 24 hours
D.7 days only
Explanation: Epicutaneous patch testing assesses type IV (delayed-type, cell-mediated) hypersensitivity. Standard protocol requires applying patches for 48 hours, followed by removal and a primary reading at 48 hours (or 30 minutes after removal to allow pressure erythema to fade), and a mandatory second reading at 72 to 96 hours (day 3 or 4) to distinguish allergic reactions (which persist or crescendo) from irritant reactions (which decrescendo). Certain allergens such as corticosteroids or neomycin may require an additional late reading at day 7.

About the Czech Dermatovenerology Board Examination Exam

The Atestační zkouška v oboru Dermatovenerologie is the statutory specialty examination for Czech specialist competence under Act No. 95/2004 Coll. and Decree No. 282/2019 Coll. The official assessment combines practical patient evaluation and interpretation of histopathology, mycology, and dermoscopy material with an oral examination before an appointed board. This independent English-language MCQ bank adapts the published curriculum for study; it is not an official translation or format simulation and does not substitute for oral or practical preparation.

Assessment

The specialty examination includes a practical patient work-up plus interpretation of four histopathology slides, three mycology samples, and three dermoscopy cases, followed by an oral theoretical examination before an appointed at least 3-member board. Both parts are evaluated pass/fail (prospěl/a).

Time Limit

No fixed total duration is published; oral preparation is at least 30 minutes, with practical assessment as required by the specialty program.

Passing Score

Pass (prospěl/a) evaluated across practical and theoretical components by unanimous or majority committee vote

Exam Fee

500 CZK for first attempt (250 CZK practical, 250 CZK theoretical); 3,500 CZK for 1st retake; 5,000 CZK for 2nd retake under Government Regulation No. 324/2018 Coll. (Ministerstvo zdravotnictví České republiky (MZ ČR) / Lékařské fakulty / Institut postgraduálního vzdělávání ve zdravotnictví (IPVZ))

Czech Dermatovenerology Board Examination Exam Content Outline

Not published

Inflammatory & Autoimmune Dermatoses

Psoriasis, lichen planus, autoimmune blistering diseases, connective tissue diseases (lupus, dermatomyositis, systemic sclerosis), severe cutaneous adverse drug reactions (SJS/TEN, DRESS, AGEP), and vasculitis.

Not published

Dermato-Oncology & Skin Neoplasms

Melanoma AJCC 8th edition staging, surgical margins, sentinel node biopsy, targeted and immune therapies, BCC subtypes and hedgehog inhibitors, cSCC, CTCL/mycosis fungoides, and soft tissue sarcomas.

Not published

Infectious Skin Diseases

Bacterial pyodermas, erysipelas vs cellulitis, necrotizing fasciitis, Lyme borreliosis stages and diagnostics, viral exanthems, superficial and deep fungal infections, and parasitic infestations (scabies, leishmaniasis).

Not published

Venereology & Sexually Transmitted Infections

Syphilis staging, serology algorithms, benzathine penicillin protocols, Czech public health surveillance (Act No. 258/2000 Coll.), gonorrhea resistance, chlamydia, LGV, chancroid, and HIV dermatoses.

Not published

Pediatric & General Dermatology / Biologics

Genodermatoses, pediatric vascular tumors/malformations, acne vulgaris, hidradenitis suppurativa, biologics (TNF, IL-17, IL-23, IL-4/13 inhibitors), small molecules (JAK inhibitors), and phototherapy.

How to Pass the Czech Dermatovenerology Board Examination Exam

What You Need to Know

  • Passing score: Pass (prospěl/a) evaluated across practical and theoretical components by unanimous or majority committee vote
  • Assessment: The specialty examination includes a practical patient work-up plus interpretation of four histopathology slides, three mycology samples, and three dermoscopy cases, followed by an oral theoretical examination before an appointed at least 3-member board. Both parts are evaluated pass/fail (prospěl/a).
  • Time limit: No fixed total duration is published; oral preparation is at least 30 minutes, with practical assessment as required by the specialty program.
  • Exam fee: 500 CZK for first attempt (250 CZK practical, 250 CZK theoretical); 3,500 CZK for 1st retake; 5,000 CZK for 2nd retake under Government Regulation No. 324/2018 Coll.

Keys to Passing

  • Work through all 100 available questions
  • Review every answer and explanation
  • Track weak areas and revisit them
  • Use our AI tutor for tough concepts

Czech Dermatovenerology Board Examination Study Tips from Top Performers

1Master the AJCC 8th edition melanoma staging system, surgical resection margins based on Breslow depth, and indications for sentinel lymph node biopsy and adjuvant immunotherapy/targeted therapy.
2Thoroughly review European and Czech guidelines on psoriasis biologic therapy, including screening for latent tuberculosis, hepatitis B/C, and choice among TNF-alpha, IL-17, and IL-23 inhibitors.
3Understand the staged serological interpretation of syphilis (screening with treponemal assays, monitoring treatment response and reinfection with non-treponemal RPR/VDRL titers, and CSF criteria for neurosyphilis).
4Be confident in differentiating autoimmune bullous diseases by cleavage level (suprabasal in pemphigus vulgaris vs subepidermal in bullous pemphigoid) and direct immunofluorescence patterns (epidermal intercellular vs linear basement membrane zone).

Frequently Asked Questions

What is the official format of the Czech Atestace in Dermatovenerologie?

The current specialty program requires a practical patient work-up plus interpretation of histopathology, mycology, and dermoscopy material, followed by an oral theoretical examination before an appointed committee of at least three members.

How does this practice bank adapt the official Czech board examination?

The official statutory examination is conducted orally and clinically in Czech. This question bank is an English-language multiple-choice study adaptation designed to reinforce diagnostic criteria, clinical pharmacology, histopathology, and evidence-based treatment algorithms corresponding to the official Věstník MZ ČR educational curriculum and guidelines from the Czech Dermatovenerological Society (ČDS ČLS JEP) and European Academy of Dermatology and Venereology (EADV).

What are the examination fees under Czech law?

Under Government Regulation No. 324/2018 Coll. (Nařízení vlády č. 324/2018 Sb.), the fee for the regular examination attempt is 500 CZK (250 CZK practical, 250 CZK theoretical). If a retake is required, the first retake fee is 3,500 CZK (2,000 CZK practical, 1,500 CZK theoretical) and the second retake fee is 5,000 CZK (3,000 CZK practical, 2,000 CZK theoretical).

What is the duration and structure of postgraduate training in Dermatovenerology?

Specialized training lasts a minimum of 5 years (60 months) according to Věstník MZ ČR. It is structured into two 30-month halves: the foundational trunk (dermatovenerologický kmen) followed by specialized dermatovenerology training, covering inpatient dermatology, pediatric dermatology, corrective dermatology, venereology, and dermatopathology.

What public health legislation governs venereology in the Czech Republic?

Venereology practice and STI reporting are regulated under Act No. 258/2000 Coll. on Public Health Protection (Zákon č. 258/2000 Sb. o ochraně veřejného zdraví). Diagnosed cases of syphilis, gonorrhea, lymphogranuloma venereum, and chancroid require mandatory statutory notification, epidemiological contact tracing (depistáž), and compulsory treatment.