100+ Free ABOM Oral Medicine Practice Questions

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Question 1

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A patient with poorly controlled type 2 diabetes is most likely to demonstrate which oral finding?

Macroglossia with scalloping

Xerostomia, increased periodontal disease, and delayed wound healing

Cinnamon-induced contact stomatitis

Bilateral Stensen duct calculi

to track

2026 Statistics

Key Facts: ABOM Oral Medicine Exam

Exam Components

Written Examination, Case Portfolio, and Oral Examination

2020

ADA Recognition

Oral medicine recognized as a dental specialty by the ADA

~15%

Oral Systemic Weight

Largest single domain on ABOM content outline

~$2,000-$2,500

2026 Exam Fee

ABOM (verify current schedule)

2 yr

Residency Requirement

CODA-accredited oral medicine residency

100

FREE Practice Questions

Covering the 2026 ABOM content outline

The ABOM Certification is a multi-component process from the American Board of Oral Medicine — Written Examination, Case Portfolio, and Oral Examination — for dentists specializing in the medical management of oral disease in medically complex patients. Content spans oral systemic manifestations (~15%), oral mucosal disorders (~12%), salivary gland (~10%), medical management (~10%), infections (~10%), pre-malignant/malignant (~10%), medically complex dental management (~8%), immune-mediated disease (~8%), burning mouth (~5%), aphthous stomatitis (~5%), TMD (~5%), diagnostic labs (~4%), and dysesthesias (~3%). Fee is approximately $2,000-$2,500; requires a CODA-accredited oral medicine residency or experience pathway. ABOM was recognized by the ADA as a dental specialty in 2020.

Sample ABOM Oral Medicine Practice Questions

Try these sample questions to test your ABOM Oral Medicine exam readiness. Each question includes a detailed explanation. Start the interactive quiz above for the full 100+ question experience with AI tutoring.

1A patient with poorly controlled type 2 diabetes is most likely to demonstrate which oral finding?

A.Macroglossia with scalloping

B.Xerostomia, increased periodontal disease, and delayed wound healing

C.Cinnamon-induced contact stomatitis

D.Bilateral Stensen duct calculi

Explanation: Uncontrolled diabetes is associated with xerostomia (reduced salivary flow), accelerated periodontitis, candidal overgrowth, burning mouth sensation, and delayed wound healing secondary to microvascular disease and impaired neutrophil function.

2Oral hairy leukoplakia is caused by which virus and classically presents where?

A.HPV 16, soft palate

B.HSV-1, attached gingiva

C.Epstein-Barr virus on the lateral tongue

D.CMV, buccal mucosa

Explanation: Oral hairy leukoplakia is an EBV-driven corrugated, non-wipeable white lesion of the lateral tongue, most often seen in HIV/AIDS and other immunosuppressed patients. It is a clinical marker of immune decline.

3A patient with AIDS presents with a reddish-purple macular lesion on the hard palate that does not blanch. The most likely diagnosis is:

A.Hematoma

B.Kaposi sarcoma

C.Pyogenic granuloma

D.Thrombocytopenic purpura

Explanation: Kaposi sarcoma (HHV-8 related) commonly presents in the oral cavity as non-blanching red-purple macules, plaques, or nodules, with the hard palate being the most frequently involved site in HIV patients.

4Linear gingival erythema in a patient with HIV is best characterized by:

A.Associated severe bone loss proportional to plaque

B.A distinct 2-3 mm erythematous band along the free gingival margin that is disproportionate to plaque

C.Deep pseudopockets without bleeding

D.Pathognomonic cratered papillae

Explanation: Linear gingival erythema appears as a vivid red band along the free gingival margin that is disproportionate to local plaque, often unresponsive to conventional hygiene. NUP (necrotizing ulcerative periodontitis) is a separate, more destructive HIV-related entity with cratered papillae.

5Gingival hyperplasia that is most suggestive of an underlying leukemia is:

A.Generalized fibrous enlargement in a patient on phenytoin

B.Diffuse, boggy, bluish-red enlargement of the marginal and attached gingiva

C.Interdental papillary recession

D.Strawberry gingivitis

Explanation: Leukemic gingival infiltration (classically acute monocytic, AML M5) produces boggy, swollen, bluish-red marginal and attached gingiva that may obscure teeth and bleed spontaneously. Strawberry gingivitis suggests granulomatosis with polyangiitis.

6A patient with severe thrombocytopenia (platelets 15,000/µL) is most likely to present intra-orally with:

A.Nodular gingival enlargement

B.Petechiae and ecchymoses on the palate with spontaneous gingival bleeding

C.Xerostomia and burning tongue

D.Hyperpigmentation of the buccal mucosa

Explanation: Thrombocytopenia causes submucosal hemorrhage that manifests as petechiae, purpura, and ecchymoses (especially on the palate) and spontaneous gingival bleeding. Elective dental procedures are typically deferred until platelets exceed ~50,000/µL.

7Plummer-Vinson syndrome is characterized by the triad of:

A.Esophageal webs, iron deficiency anemia, and atrophic glossitis

B.Pernicious anemia, Hunter glossitis, and neuropathy

C.Dysphagia, leukoplakia, and gastric ulcers

D.Vitiligo, candidiasis, and hypoparathyroidism

Explanation: Plummer-Vinson (Paterson-Brown-Kelly) syndrome: iron deficiency anemia, dysphagia from upper esophageal webs, and atrophic glossitis/angular cheilitis. It carries increased risk for upper aerodigestive squamous cell carcinoma.

8A beefy red, smooth, painful tongue in a patient with macrocytic anemia and paresthesias suggests:

A.Median rhomboid glossitis

B.Iron deficiency anemia

C.Vitamin B12 deficiency (Hunter glossitis)

D.Geographic tongue

Explanation: B12 (cobalamin) deficiency causes atrophic 'bald' glossitis (Hunter or Moeller glossitis) with burning and pain, often accompanied by macrocytic anemia and subacute combined degeneration. Replacement with parenteral B12 reverses mucosal findings.

9Which oral finding is classically associated with systemic lupus erythematosus?

A.Radial perioral furrowing with microstomia

B.Central erythematous plaque with radiating white striae on the palate

C.Bilateral parotid lipomatosis

D.Macroglossia with waxy deposits

Explanation: SLE oral lesions typically show a central erythematous or ulcerated zone with peripheral radiating white striae (discoid-like), often on the palate. Scleroderma produces radial perioral furrows and microstomia; amyloid causes macroglossia.

10A patient with scleroderma is most likely to demonstrate which oral/radiographic finding?

A.Widened periodontal ligament space on radiographs

B.Focal enamel pitting

C.Tori of the mandible

D.Multiple supernumerary teeth

Explanation: Classic scleroderma orofacial findings include microstomia, radial perioral furrowing, xerostomia (if secondary Sjögren), and a uniformly widened periodontal ligament space (especially around posterior teeth) on imaging due to collagen deposition.

About the ABOM Oral Medicine Exam

The American Board of Oral Medicine (ABOM) Certification, recognized by the American Dental Association as a dental specialty in 2020, validates expertise in the non-surgical medical management of oral and maxillofacial conditions in medically complex patients. The certification process comprises a Written Examination, a Case Portfolio demonstrating breadth of oral medicine practice, and an Oral Examination. Content spans oral mucosal disorders (lichen planus, pemphigus vulgaris with Nikolsky sign and anti-desmoglein, mucous membrane pemphigoid), salivary gland disease (Sjogren syndrome with anti-SSA/SSB, sialolithiasis, IgG4-related disease), oral manifestations of systemic disease (GI, hematologic, endocrine, HIV, vitamin deficiency), oral infections (candidiasis, HSV-1/VZV/EBV/HPV), pre-malignant and malignant lesions (leukoplakia, erythroplakia, oral SCC), medical management (AHA 2007/2021 IE prophylaxis, anticoagulation, MRONJ AAOMS 2022), burning mouth syndrome, recurrent aphthous stomatitis, TMD/orofacial pain, and diagnostic laboratory medicine. Requires completion of a CODA-accredited oral medicine residency (minimum 2 years) or a documented experience pathway.

Questions

100 scored questions

Time Limit

Multi-component: Written Examination, Case Portfolio, and Oral Examination on separate dates

Passing Score

Criterion-referenced scaled score set by ABOM (modified Angoff standard)

Exam Fee

~$2,000-$2,500 examination fee (ABOM 2026 — verify current schedule) (American Board of Oral Medicine (ABOM) / American Academy of Oral Medicine (AAOM))

ABOM Oral Medicine Exam Content Outline

~15%

Oral Systemic Manifestations

Oral signs of systemic disease: GI (Crohn cobblestoning, ulcerative colitis pyostomatitis vegetans, celiac aphthae), hematologic (leukemia gingival hypertrophy, iron-deficiency atrophic glossitis Plummer-Vinson, thrombocytopenia petechiae/ecchymoses), endocrine (diabetes — candidiasis and periodontitis; Addison hyperpigmentation; hypothyroid macroglossia), renal (uremic stomatitis), vitamin deficiency (B12/folate glossitis, scurvy bleeding gingiva, B2 angular cheilitis), HIV (hairy leukoplakia EBV, Kaposi HHV-8, linear gingival erythema, candidiasis), amyloidosis (macroglossia with indentations), sarcoidosis (Heerfordt — parotid/uveitis/facial palsy; labial minor salivary gland biopsy non-caseating granulomas).

~12%

Oral Mucosal Disorders

Oral lichen planus (reticular with Wickham striae most common; erosive/atrophic symptomatic; T-cell mediated; topical clobetasol first-line; ~1% malignant transformation risk for erosive), lichenoid drug reactions (ACEi, NSAIDs, hypoglycemics, antimalarials) and contact reactions (amalgam), geographic tongue (benign migratory glossitis — reassurance), fissured tongue, leukoedema (bilateral, disappears on stretch), white sponge nevus (keratin 4/13), morsicatio buccarum, frictional keratosis, nicotine stomatitis (palatal), actinic cheilitis (premalignant), median rhomboid glossitis (candidiasis).

~10%

Salivary Gland Disorders

Sialolithiasis (submandibular Wharton duct most common due to viscous mucinous secretions and upward drainage), sialadenitis (acute suppurative S. aureus, chronic recurrent parotitis), sialadenosis (bilateral parotid in diabetes/alcoholism/bulimia), mucocele and ranula (sublingual — plunging), necrotizing sialometaplasia (self-healing palatal ulcer mimicking SCC), IgG4-related disease (Mikulicz), Sjogren syndrome (primary and secondary — anti-SSA/Ro, anti-SSB/La, labial minor salivary gland biopsy focus score ≥1/4 mm², ACR/EULAR 2016 criteria), xerostomia (radiation, medications — anticholinergics, Sjogren), sialendoscopy, pilocarpine and cevimeline (muscarinic M3 agonists).

~10%

Medical Management & Pharmacology

Topical and systemic corticosteroids (clobetasol 0.05%, dexamethasone rinse, triamcinolone Orabase, intralesional triamcinolone; systemic prednisone taper), calcineurin inhibitors (topical tacrolimus 0.1%, cyclosporine), immunosuppressants (azathioprine, methotrexate, mycophenolate), biologics (rituximab anti-CD20 for pemphigus), antifungals (nystatin swish-and-swallow, clotrimazole troches, fluconazole; CYP3A4 drug interactions with warfarin, statins, benzodiazepines), antivirals (acyclovir, valacyclovir, famciclovir for HSV/VZV), neuropathic pain agents (gabapentin, pregabalin, TCAs nortriptyline/amitriptyline, SNRIs duloxetine, clonazepam for BMS), topical lidocaine, magic mouthwash.

~10%

Oral Infections

Candidiasis (pseudomembranous thrush wipes off; erythematous/atrophic; angular cheilitis; chronic hyperplastic — biopsy to rule out dysplasia; median rhomboid glossitis; denture stomatitis; C. albicans most common; C. glabrata/krusei fluconazole-resistant — echinocandin), HSV-1 primary herpetic gingivostomatitis (systemic illness, keratinized and non-keratinized mucosa) and recurrent herpes labialis/intraoral recurrent (keratinized only — acyclovir, valacyclovir), VZV (herpes zoster V1/V2/V3 dermatome; Ramsay Hunt syndrome — geniculate ganglion, facial palsy with ear vesicles), EBV (hairy leukoplakia HIV, Burkitt, NPC), HPV (types 6/11 papilloma/condyloma; type 16 oropharyngeal SCC), coxsackievirus (herpangina, hand-foot-mouth), deep fungal (histoplasmosis chronic ulcer, mucormycosis diabetic ketoacidosis).

~10%

Pre-malignant & Malignant Lesions

Oral potentially malignant disorders: leukoplakia (homogeneous vs non-homogeneous — nodular/verrucous/speckled higher risk; proliferative verrucous leukoplakia highest transformation rate ~60-100%), erythroplakia (highest single-lesion transformation — biopsy every red lesion), oral submucous fibrosis (areca/betel nut), oral lichen planus (erosive 1%). Epithelial dysplasia grading mild/moderate/severe/CIS. Oral SCC (lateral tongue most common; risk factors tobacco, alcohol synergy, HPV-16 oropharyngeal; TNM AJCC 8), verrucous carcinoma (Ackerman, exophytic, low metastatic potential), adjunctive diagnostics (toluidine blue, VELscope, brush biopsy — incisional biopsy remains gold standard).

~8%

Dental Management of Medically Complex Patients

Cardiovascular: AHA 2007/2021 IE prophylaxis indications — prosthetic valves/materials, prior IE, selected CHD (unrepaired cyanotic, repaired with residual defects, within 6 mo of repair), cardiac transplant with valvulopathy; amoxicillin 2 g PO 30-60 min before; clindamycin no longer recommended (cefalexin/azithromycin alternatives). ACS precautions, hypertension pre-op thresholds, warfarin (INR), DOACs. Diabetes (HbA1c, morning appointments, hypoglycemia management). Bleeding disorders (hemophilia factor replacement, vWD desmopressin, thrombocytopenia). Chemo/radiation (mucositis, xerostomia, osteoradionecrosis). Pregnancy (second trimester ideal, left lateral positioning, radiograph shielding).

~8%

Immune-Mediated & Autoimmune Disease

Pemphigus vulgaris (IgG autoantibodies against desmoglein 3 ± 1; positive Nikolsky sign; intraepithelial acantholysis with tombstone basal cells; DIF shows fishnet intercellular IgG/C3; systemic steroids + rituximab or steroid-sparing agents), mucous membrane pemphigoid (anti-BP180/BP230; subepithelial split; DIF linear IgG/C3 at BMZ; ocular scarring symblepharon risk), bullous pemphigoid, erythema multiforme (HSV-triggered; target lesions; lip crusting) and SJS/TEN (drug-induced — sulfa, anticonvulsants, allopurinol; >30% epidermal detachment = TEN), DLE/SLE oral lesions, Behcet (oral + genital ulcers + ocular — pathergy, HLA-B51), Wegener GPA (strawberry gingivitis — c-ANCA/PR3), Kawasaki disease.

~5%

Burning Mouth Syndrome & Orofacial Pain

Primary (idiopathic) burning mouth syndrome: peri/postmenopausal women, bilateral symmetric burning pain of tongue tip/anterior palate/lip without mucosal changes, often with dysgeusia and subjective xerostomia; small-fiber neuropathic etiology proposed; management — topical clonazepam (swish-and-spit), systemic clonazepam, gabapentin, TCAs (nortriptyline), alpha-lipoic acid, CBT. Secondary burning causes (must exclude before diagnosing primary): candidiasis, xerostomia, nutritional (B12/folate/iron/zinc), diabetes, GERD, drug-induced, parafunction, contact allergy (SLS, cinnamon).

~5%

Recurrent Aphthous Stomatitis

RAS classification: minor (<1 cm, non-keratinized mucosa, heals ~7-14 d without scarring — most common), major (>1 cm, >2 weeks, scarring), herpetiform (crops of small ulcers fusing; non-keratinized mucosa). Triggers: stress, trauma, SLS toothpaste, hormones, food hypersensitivity (gluten, chocolate). Associated conditions: celiac, IBD, Behcet, HIV, cyclic neutropenia, MAGIC syndrome, PFAPA. Workup for severe/recurrent: CBC, B12, folate, iron/ferritin, celiac serologies. Management: topical corticosteroids (clobetasol, triamcinolone), amlexanox, tetracycline/doxycycline rinse, colchicine, pentoxifylline, thalidomide (HIV-associated severe).

~5%

TMD & Orofacial Pain (Brief)

Temporomandibular disorders (myofascial pain most common; disc displacement with reduction — clicking; disc displacement without reduction — closed lock; DJD/osteoarthritis), trigeminal neuralgia (carbamazepine first-line; oxcarbazepine alternative; MVD — Jannetta microvascular decompression), post-herpetic neuralgia (gabapentin, pregabalin, TCAs, topical lidocaine/capsaicin), persistent idiopathic facial pain (atypical facial pain), atypical odontalgia, occipital neuralgia, cluster headache, giant cell arteritis (>50, jaw claudication, ESR >50, temporal artery biopsy; emergency IV steroids to prevent blindness).

~4%

Diagnostic Testing & Laboratory Medicine

CBC with differential, CMP, HbA1c, coagulation (PT/INR, aPTT, platelet count/function), autoimmune serologies (ANA, anti-dsDNA, anti-Ro/SSA, anti-La/SSB, anti-desmoglein 3/1 ELISA, anti-BP180/BP230, c-ANCA/PR3, p-ANCA/MPO, RF, anti-CCP), inflammatory markers (ESR, CRP), vitamin panels (B12, folate, iron/TIBC/ferritin, 25-OH vitamin D, zinc), HIV testing, biopsy technique (incisional vs excisional, punch, brush cytology limitations), direct immunofluorescence on fresh tissue in Michel solution and indirect IF on patient serum, salt-split skin for MMP vs EBA, microbial culture and PCR.

~3%

Dysesthesias & Neurosensory Disorders

Taste disorders (ageusia, hypogeusia, dysgeusia — zinc deficiency, drug-induced — metronidazole, ACEi, chemotherapy; post-viral including COVID-19, post-radiation), smell disorders (anosmia — post-COVID, head trauma with cribriform plate injury), phantom taste, oral dysesthesia, post-surgical neurosensory deficits (inferior alveolar and lingual nerve injury after third molar surgery — paresthesia/dysesthesia/anesthesia; Seddon and Sunderland classification), trigeminal neuropathy.

How to Pass the ABOM Oral Medicine Exam

What You Need to Know

Passing score: Criterion-referenced scaled score set by ABOM (modified Angoff standard)
Exam length: 100 questions
Time limit: Multi-component: Written Examination, Case Portfolio, and Oral Examination on separate dates
Exam fee: ~$2,000-$2,500 examination fee (ABOM 2026 — verify current schedule)

Keys to Passing

Complete 500+ practice questions
Score 80%+ consistently before scheduling
Focus on highest-weighted sections
Use our AI tutor for tough concepts

ABOM Oral Medicine Study Tips from Top Performers

1Pemphigus vs Pemphigoid is a must-know discriminator. Pemphigus vulgaris: IgG against desmoglein 3 (± 1), intraepithelial split with tombstone basal cells, positive Nikolsky, DIF fishnet/chickenwire intercellular IgG/C3; treat with systemic steroids + rituximab. Mucous membrane pemphigoid: anti-BP180/BP230, subepithelial split, DIF linear IgG/C3 at basement membrane zone; high risk of ocular symblepharon and scarring — mandatory ophthalmology referral.

2Sjogren syndrome diagnosis (ACR/EULAR 2016): labial minor salivary gland biopsy showing focus score ≥1 focus of ≥50 lymphocytes per 4 mm² (weight 3), anti-SSA/Ro positive (weight 3), ocular staining score ≥5 or van Bijsterveld ≥4 (weight 1), Schirmer ≤5 mm/5 min (weight 1), unstimulated whole salivary flow ≤0.1 mL/min (weight 1). Score ≥4 meets criteria. Xerostomia management: sugar-free lozenges, xylitol, pilocarpine 5 mg QID or cevimeline 30 mg TID (M3 muscarinic agonists — contraindicated in uncontrolled asthma, narrow-angle glaucoma).

3MRONJ AAOMS 2022 staging: Stage 0 (non-exposed disease), Stage 1 (asymptomatic exposed bone), Stage 2 (symptomatic with infection), Stage 3 (extraoral fistula, pathologic fracture, extension beyond alveolar bone). Risk factors: IV bisphosphonates (zoledronate) > oral; denosumab (RANKL inhibitor); antiangiogenics. Duration of exposure and potency drive risk. Prevention: dental screening and extractions before bisphosphonate initiation; avoid elective dentoalveolar surgery on high-risk patients. Conservative treatment for early stages; surgical debridement for Stage 2/3.

4AHA 2007/2021 IE prophylaxis — indications: prosthetic cardiac valve or prosthetic material, previous infective endocarditis, certain congenital heart disease (unrepaired cyanotic CHD; repaired CHD with prosthetic material within 6 months; repaired CHD with residual defects at or adjacent to prosthetic material), cardiac transplant recipients with valvulopathy. Regimen: amoxicillin 2 g PO 30-60 min before procedure (children 50 mg/kg). Penicillin allergy: cefalexin 2 g or azithromycin/clarithromycin 500 mg (clindamycin 2021 update no longer recommended due to C. difficile and adverse events). Only for dental procedures involving gingival manipulation, periapical region, or mucosal perforation.

5Burning Mouth Syndrome is a DIAGNOSIS OF EXCLUSION. Before diagnosing primary BMS, systematically rule out: candidiasis (smear/culture), xerostomia (sialometry), nutritional deficiencies (B12, folate, iron/ferritin, zinc), diabetes (HbA1c), GERD, contact allergy (patch test for SLS, cinnamon aldehyde, metals), parafunction, medications (ACEi cause taste/burning). Once secondary causes excluded, primary BMS management: topical clonazepam (dissolve 1 mg in mouth for 3 min, spit; BID-TID), systemic clonazepam 0.25-0.5 mg, gabapentin 300-900 mg/day, nortriptyline 10-50 mg qhs, alpha-lipoic acid 600 mg/day, CBT.

Frequently Asked Questions

What is the ABOM Oral Medicine Certification?

The American Board of Oral Medicine (ABOM) Certification validates expertise in the non-surgical medical management of oral and maxillofacial disease in medically complex patients. Oral medicine was recognized by the American Dental Association as a dental specialty in 2020. The certification process comprises a Written Examination, a Case Portfolio demonstrating breadth of oral medicine practice, and an Oral Examination — all administered by ABOM in partnership with the American Academy of Oral Medicine (AAOM).

Who is eligible to sit for ABOM certification?

Candidates must hold a D.D.S., D.M.D., or equivalent dental degree with an unrestricted dental license and complete a CODA-accredited oral medicine residency of at least 2 years. An alternative experience pathway is available for dentists with significant documented clinical and scholarly activity in oral medicine per ABOM eligibility requirements. Program director attestation and a Case Portfolio demonstrating breadth of practice are required.

What is the format of ABOM certification?

ABOM certification is a multi-component process: (1) Written Examination — computer-based multiple-choice test covering the oral medicine content outline; (2) Case Portfolio — submission of clinical cases spanning the breadth of oral medicine practice; (3) Oral Examination — case-based interview with examiners. Components are administered on separate dates. Successful completion of the Written Examination is required before progression to subsequent components.

How much does ABOM certification cost in 2026?

The 2026 ABOM examination fee is approximately $2,000-$2,500 — verify the current schedule on the ABOM website. Additional fees apply for Case Portfolio review and the Oral Examination. Ongoing continuing certification fees apply after initial certification. Cancellation and refund policies follow ABOM policy with decreasing refunds as the exam date approaches.

When is the 2026 exam administered?

ABOM administers its Written Examination, Case Portfolio review, and Oral Examination on published dates each year, typically with Written Examination offered annually or biennially and the Case Portfolio/Oral Examination scheduled in association with AAOM meetings. Exact 2026 dates and deadlines should be confirmed on the ABOM website.

How is the exam scored?

ABOM uses criterion-referenced scaled scoring with a passing standard set by subject-matter experts using the modified Angoff method. A candidate's pass/fail result depends on performance relative to the fixed cut-score, not on other candidates. The Case Portfolio and Oral Examination are graded against published ABOM rubrics. All components must be passed for Diplomate status.

What are the highest-yield topics?

Highest-yield topics include oral lichen planus and lichenoid reactions, pemphigus vulgaris (Nikolsky, anti-desmoglein, DIF intercellular), mucous membrane pemphigoid, Sjogren syndrome (anti-SSA/SSB, focus score, ACR/EULAR), MRONJ AAOMS 2022 staging, AHA 2007/2021 IE prophylaxis regimens (amoxicillin 2 g), candidiasis and antifungal selection (fluconazole vs echinocandins for resistant Candida), HSV/VZV/HPV oral manifestations, oral potentially malignant disorders (PVL, erythroplakia), burning mouth syndrome management (clonazepam, gabapentin, TCAs), and aphthous stomatitis workup.

How should I study for this exam?

Use a structured 12-18 month plan during and after oral medicine residency. Map to the ABOM content outline: begin with oral mucosal disease and autoimmune disease (DIF patterns), then salivary gland and Sjogren, infections, oral oncology and MRONJ, medical management of medically complex patients, pain and BMS/RAS, and diagnostic laboratory medicine. Integrate primary textbooks (Burket's Oral Medicine, Glick; Scully Oral and Maxillofacial Medicine), AAOM clinicians' guides, and current AAOMS and AHA guidelines. Build your Case Portfolio concurrently — do not delay case documentation.