Key Takeaways

  • The central nervous system (CNS) consists of the brain and spinal cord; the peripheral nervous system (PNS) includes all nerves outside the CNS
  • Upper motor neuron (UMN) lesions present with spasticity, hyperreflexia, clonus, positive Babinski sign, and no muscle atrophy initially
  • Lower motor neuron (LMN) lesions present with flaccidity, hyporeflexia/areflexia, fasciculations, and rapid muscle atrophy
  • Dermatomes are areas of skin supplied by a single spinal nerve root: C5 (lateral arm), C6 (thumb), C7 (middle finger), T4 (nipple line), T10 (umbilicus), L4 (medial leg), L5 (dorsal foot), S1 (lateral foot)
  • Myotomes represent the muscles innervated by a single nerve root: C5 (deltoid), C6 (biceps/wrist extensors), C7 (triceps/wrist flexors), L3 (quadriceps), L4 (tibialis anterior), L5 (extensor hallucis longus), S1 (gastrocnemius)
  • The corticospinal tract (lateral) is the primary motor pathway; damage causes contralateral UMN signs below the level of lesion
  • The dorsal columns (posterior) carry proprioception, vibration, and fine touch; damage causes ipsilateral loss of these sensations
  • The spinothalamic tract carries pain and temperature; damage causes contralateral loss of pain and temperature sensation
Last updated: February 2026

Neuroanatomy & Neurophysiology

Understanding the structure and function of the nervous system is essential for the NPTE neuromuscular domain. Physical therapists must be able to localize neurological lesions, predict functional deficits, and select appropriate interventions.


CNS vs. PNS Overview

FeatureCentral Nervous System (CNS)Peripheral Nervous System (PNS)
ComponentsBrain and spinal cordCranial nerves, spinal nerves, peripheral nerves
ProtectionSkull, vertebrae, meninges, CSFEpineurium, perineurium, endoneurium
RegenerationVery limited (glial scarring inhibits regrowth)Can regenerate at ~1 mm/day (1 inch/month)
MyelinationOligodendrocytesSchwann cells

Upper Motor Neuron (UMN) vs. Lower Motor Neuron (LMN) Lesions

This is one of the most tested concepts on the NPTE. UMN and LMN lesions produce opposite clinical presentations:

FeatureUMN LesionLMN Lesion
Muscle toneSpasticity (increased)Flaccidity (decreased)
Deep tendon reflexesHyperreflexia (increased)Hyporeflexia or areflexia (decreased/absent)
Babinski signPositive (toes fan, great toe extends)Negative (normal flexor response)
ClonusMay be presentAbsent
Muscle atrophyMinimal initially (disuse atrophy over time)Rapid, significant atrophy (denervation)
FasciculationsAbsentPresent (visible muscle twitching)
DistributionAffects groups of muscles (patterns)Affects individual muscles (segmental)
ExamplesStroke, TBI, MS, SCI (above conus)Peripheral neuropathy, radiculopathy, Guillain-Barre, SCI (at level of injury)

Key UMN Spasticity Pattern:

  • Upper extremity: Flexor pattern (shoulder adduction/IR, elbow flexion, forearm pronation, wrist/finger flexion)
  • Lower extremity: Extensor pattern (hip extension/adduction/IR, knee extension, ankle plantarflexion/inversion)

Spinal Cord Tracts

Understanding the major ascending (sensory) and descending (motor) tracts allows localization of spinal cord lesions:

Major Ascending (Sensory) Tracts

TractLocationModalityCrosses (Decussation)
Dorsal columns (posterior)Posterior spinal cordProprioception, vibration, fine/discriminative touchMedulla (crosses high)
Spinothalamic (lateral)Anterolateral spinal cordPain and temperatureSpinal cord level (crosses at entry)
Anterior spinothalamicAnterior spinal cordCrude touch, pressureSpinal cord level

Major Descending (Motor) Tract

TractLocationFunctionCrosses
Lateral corticospinalLateral spinal cordVoluntary motor control (skilled movements)Medulla (pyramidal decussation)

Clinical Significance of Decussation:

  • Dorsal columns cross in the medulla → Damage in the spinal cord causes ipsilateral loss of proprioception and vibration
  • Spinothalamic tract crosses at spinal cord level → Damage in the spinal cord causes contralateral loss of pain and temperature
  • Corticospinal tract crosses in the medulla → Damage above the decussation (brain) causes contralateral motor deficits; damage below (spinal cord) causes ipsilateral motor deficits

Dermatomes and Myotomes

Key Dermatome Landmarks

Nerve RootSensory Area
C5Lateral arm (deltoid patch)
C6Lateral forearm, thumb, index finger
C7Middle finger
C8Ring finger, little finger, medial forearm
T4Nipple line
T10Umbilicus
L1Inguinal ligament region
L3Anterior thigh above knee
L4Medial leg
L5Dorsum of foot, first web space
S1Lateral foot, small toe
S2-S4Saddle region (perianal area)

Key Myotome Testing

Nerve RootPrimary Muscle/Action
C5Deltoid — shoulder abduction
C6Biceps — elbow flexion; wrist extensors
C7Triceps — elbow extension; wrist flexors
C8Finger flexors — grip strength
T1Hand intrinsics — finger abduction
L2Iliopsoas — hip flexion
L3Quadriceps — knee extension
L4Tibialis anterior — ankle dorsiflexion
L5Extensor hallucis longus — great toe extension
S1Gastrocnemius/soleus — ankle plantarflexion
S2Hamstrings — knee flexion
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Spinal Cord Cross-Section: Major Tracts
Test Your Knowledge

A patient presents with spasticity, hyperreflexia, and a positive Babinski sign. This presentation is MOST consistent with:

A
B
C
D
Test Your Knowledge

Damage to the dorsal columns of the spinal cord at T6 would result in loss of which sensation BELOW the level of injury?

A
B
C
D
Test Your KnowledgeMatching

Match each dermatome to its corresponding sensory area.

Match each item on the left with the correct item on the right

1
C6
2
T4
3
T10
4
L5
5
S1
Test Your Knowledge

The L4 myotome is best tested by assessing which muscle action?

A
B
C
D
Test Your KnowledgeFill in the Blank

Peripheral nerves can regenerate at a rate of approximately _____ per day (or about 1 inch per month).

Type your answer below

Test Your Knowledge

A patient with an UMN lesion would typically exhibit which posture in the affected upper extremity?

A
B
C
D